p66Shc signaling and autophagy impact on C2C12 myoblast differentiation during senescence

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Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: 1 Online-Ressource.

Language: Englisch

Bibliographic citation: p66Shc signaling and autophagy impact on C2C12 myoblast differentiation during senescence ; volume:15 ; number:3 ; day:8 ; month:3 ; year:2024 ; pages:1-16 ; date:3.2024
Cell death & disease ; 15, Heft 3 (8.3.2024), 1-16, 3.2024

Creator: Potes, Yaiza
Bermejo-Millo, Juan C.
Mendes, Catarina
Castelão-Baptista, José P.
Díaz-Luis, Andrea
Pérez-Martínez, Zulema
Solano, Juan J.
Sardão, Vilma A.
Oliveira, Paulo J.
Caballero, Beatriz
Coto-Montes, Ana
Vega-Naredo, Ignacio

Contributor: SpringerLink (Online service)

DOI: 10.1038/s41419-024-06582-0

URN: urn:nbn:de:101:1-2405112128208.040964458915

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:47 AM CEST

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Potes, Yaiza
Bermejo-Millo, Juan C.
Mendes, Catarina
Castelão-Baptista, José P.
Díaz-Luis, Andrea
Pérez-Martínez, Zulema
Solano, Juan J.
Sardão, Vilma A.
Oliveira, Paulo J.
Caballero, Beatriz
Coto-Montes, Ana
Vega-Naredo, Ignacio
SpringerLink (Online service)

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p66Shc signaling and autophagy impact on C2C12 myoblast differentiation during senescence

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Other Objects (12)

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Elevation of phosphate levels impairs skeletal myoblast differentiation

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MiR-16-5p targets SESN1 to regulate the p53 signaling pathway, affecting myoblast proliferation and apoptosis, and is involved in myoblast differentiation

Continuous exposure of isoprenaline inhibits myoblast differentiation and fusion through PKA/ERK1/2-FOXO1 signaling pathway

2-D08 treatment regulates C2C12 myoblast proliferation and differentiation via the Erk1/2 and proteasome signaling pathways

Knockdown of subunit 3 of the COP9 signalosome inhibits C2C12 myoblast differentiation via NF-KappaB signaling pathway

Targeting myomiRs by tocotrienol-rich fraction to promote myoblast differentiation

Collagen XXV promotes myoblast fusion during myogenic differentiation and muscle formation

BVES is a novel interactor of ANO5 and regulates myoblast differentiation

Continuous exposure to isoprenaline reduced myotube size by delaying myoblast differentiation and fusion through the NFAT-MEF2C signaling pathway

Requirement for PINCH in skeletal myoblast differentiation

Slc2a6 regulates myoblast differentiation by targeting LDHB

Elevation of phosphate levels impairs skeletal myoblast differentiation

PGC-1α is associated with C2C12 Myoblast differentiation

MiR-16-5p targets SESN1 to regulate the p53 signaling pathway, affecting myoblast proliferation and apoptosis, and is involved in myoblast differentiation

Continuous exposure of isoprenaline inhibits myoblast differentiation and fusion through PKA/ERK1/2-FOXO1 signaling pathway

2-D08 treatment regulates C2C12 myoblast proliferation and differentiation via the Erk1/2 and proteasome signaling pathways

Knockdown of subunit 3 of the COP9 signalosome inhibits C2C12 myoblast differentiation via NF-KappaB signaling pathway

Targeting myomiRs by tocotrienol-rich fraction to promote myoblast differentiation

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BVES is a novel interactor of ANO5 and regulates myoblast differentiation

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Requirement for PINCH in skeletal myoblast differentiation

Slc2a6 regulates myoblast differentiation by targeting LDHB

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PGC-1α is associated with C2C12 Myoblast differentiation

MiR-16-5p targets SESN1 to regulate the p53 signaling pathway, affecting myoblast proliferation and apoptosis, and is involved in myoblast differentiation

Continuous exposure of isoprenaline inhibits myoblast differentiation and fusion through PKA/ERK1/2-FOXO1 signaling pathway

2-D08 treatment regulates C2C12 myoblast proliferation and differentiation via the Erk1/2 and proteasome signaling pathways

Knockdown of subunit 3 of the COP9 signalosome inhibits C2C12 myoblast differentiation via NF-KappaB signaling pathway

Targeting myomiRs by tocotrienol-rich fraction to promote myoblast differentiation

Collagen XXV promotes myoblast fusion during myogenic differentiation and muscle formation

BVES is a novel interactor of ANO5 and regulates myoblast differentiation

Continuous exposure to isoprenaline reduced myotube size by delaying myoblast differentiation and fusion through the NFAT-MEF2C signaling pathway

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