Overexpression of SGLT2 in the kidney of a P. gingivalis LPS-induced diabetic nephropathy mouse model
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1471-2369
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Overexpression of SGLT2 in the kidney of a P. gingivalis LPS-induced diabetic nephropathy mouse model ; volume:22 ; number:1 ; day:23 ; month:8 ; year:2021 ; pages:1-10 ; date:12.2021
BMC nephrology ; 22, Heft 1 (23.8.2021), 1-10, 12.2021
- Creator
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Kajiwara, Koichiro
Sawa, Yoshihiko
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/s12882-021-02506-8
- URN
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urn:nbn:de:101:1-2021110820443340375654
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:37 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Kajiwara, Koichiro
- Sawa, Yoshihiko
- SpringerLink (Online service)
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Immunohistochemical study for the expression of leukocyte adhesion molecules, and FGF23 and ACE2 in P. gingivalis LPS-induced diabetic nephropathy
The promotion of nephropathy by Porphyromonas gingivalis lipopolysaccharide via toll-like receptors
Amelioration of diabetic nephropathy by SGLT2 inhibitors independent of its glucose-lowering effect: A possible role of SGLT2 in mesangial cells
SGLT2 and cancer
A new subtype of Entamoeba gingivalis: “E. gingivalis ST2, kamaktli variant”
Systemic LPS-induced Aβ-solubilization and clearance in AβPP-transgenic mice is diminished by heparanase overexpression
Overexpression of DEL-1 Downregulates SH3BP2 Expression and Inhibits Porphyromonas gingivalis-induced Gingival Inflammation In Vivo and In Vitro
LncRNA GAS5 Overexpression Reverses LPS-Induced Inflammatory Injury and Apoptosis Through Up-Regulating KLF2 Expression in ATDC5 Chondrocytes
SGLT2-Hemmer : Geringere Mortalität und Nierenschutz
Direct cardiac effects of SGLT2 inhibitors
Targeting high glucose-induced epigenetic modifications at cardiac level: the role of SGLT2 and SGLT2 inhibitors
A Fluorescent Glucose Transport Assay for Screening SGLT2 Inhibitors in Endogenous SGLT2-Expressing HK-2 Cells
Immunohistochemical study for the expression of leukocyte adhesion molecules, and FGF23 and ACE2 in P. gingivalis LPS-induced diabetic nephropathy
The promotion of nephropathy by Porphyromonas gingivalis lipopolysaccharide via toll-like receptors
Amelioration of diabetic nephropathy by SGLT2 inhibitors independent of its glucose-lowering effect: A possible role of SGLT2 in mesangial cells
SGLT2 and cancer
A new subtype of Entamoeba gingivalis: “E. gingivalis ST2, kamaktli variant”
Systemic LPS-induced Aβ-solubilization and clearance in AβPP-transgenic mice is diminished by heparanase overexpression
Overexpression of DEL-1 Downregulates SH3BP2 Expression and Inhibits Porphyromonas gingivalis-induced Gingival Inflammation In Vivo and In Vitro
LncRNA GAS5 Overexpression Reverses LPS-Induced Inflammatory Injury and Apoptosis Through Up-Regulating KLF2 Expression in ATDC5 Chondrocytes
SGLT2-Hemmer : Geringere Mortalität und Nierenschutz
Direct cardiac effects of SGLT2 inhibitors
Targeting high glucose-induced epigenetic modifications at cardiac level: the role of SGLT2 and SGLT2 inhibitors