Kir4.1 channels contribute to astrocyte CO2/H+-sensitivity and the drive to breathe

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Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: 1 Online-Ressource.

Language: Englisch

Bibliographic citation: Kir4.1 channels contribute to astrocyte CO2/H+-sensitivity and the drive to breathe ; volume:7 ; number:1 ; day:28 ; month:3 ; year:2024 ; pages:1-13 ; date:12.2024
Communications biology ; 7, Heft 1 (28.3.2024), 1-13, 12.2024

Creator: Cleary, Colin M.
Browning, Jack L.
Armbruster, Moritz
Sobrinho, Cleyton R.
Strain, Monica L.
Jahanbani, Sarvin
Soto-Perez, Jaseph
Hawkins, Virginia E.
Dulla, Chris G.
Olsen, Michelle L.
Mulkey, Daniel K.

Contributor: SpringerLink (Online service)

DOI: 10.1038/s42003-024-06065-0

URN: urn:nbn:de:101:1-2406122110445.348396492625

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:45 AM CEST

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Associated

Cleary, Colin M.
Browning, Jack L.
Armbruster, Moritz
Sobrinho, Cleyton R.
Strain, Monica L.
Jahanbani, Sarvin
Soto-Perez, Jaseph
Hawkins, Virginia E.
Dulla, Chris G.
Olsen, Michelle L.
Mulkey, Daniel K.
SpringerLink (Online service)

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Kir4.1 channels contribute to astrocyte CO2/H+-sensitivity and the drive to breathe

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Other Objects (12)

Cx43 hemichannels and panx1 channels contribute to ethanol-induced astrocyte dysfunction and damage

TRPM8 and TRPA1 do not contribute to dental pulp sensitivity to cold

Cavβ surface charged residues contribute to the regulation of neuronal calcium channels

Heme molecule functions as an endogenous agonist of astrocyte TLR2 to contribute to secondary brain damage after intracerebral hemorrhage

Both local stability and dispersal contribute to metacommunity sensitivity to asynchronous habitat availability

MiR-136 modulates glioma cell sensitivity to temozolomide by targeting astrocyte elevated gene-1

Channels and Transporters in Astrocyte Volume Regulation in Health and Disease

BK channels promote action potential repolarization in skeletal muscle but contribute little to myotonia

Imbalance and dysfunction of transient receptor potential channels contribute to the pathogenesis of hypertension

Specific ion channels contribute to key elements of pathology during secondary degeneration following neurotrauma

Overexpression of CPEB3 leads to astrocyte dysfunction

Astrocyte heterogeneity: from brain injury to repair

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TRPM8 and TRPA1 do not contribute to dental pulp sensitivity to cold

Cavβ surface charged residues contribute to the regulation of neuronal calcium channels

Heme molecule functions as an endogenous agonist of astrocyte TLR2 to contribute to secondary brain damage after intracerebral hemorrhage

Both local stability and dispersal contribute to metacommunity sensitivity to asynchronous habitat availability

MiR-136 modulates glioma cell sensitivity to temozolomide by targeting astrocyte elevated gene-1

Channels and Transporters in Astrocyte Volume Regulation in Health and Disease

BK channels promote action potential repolarization in skeletal muscle but contribute little to myotonia

Imbalance and dysfunction of transient receptor potential channels contribute to the pathogenesis of hypertension

Specific ion channels contribute to key elements of pathology during secondary degeneration following neurotrauma

Overexpression of CPEB3 leads to astrocyte dysfunction

Astrocyte heterogeneity: from brain injury to repair

Cx43 hemichannels and panx1 channels contribute to ethanol-induced astrocyte dysfunction and damage

TRPM8 and TRPA1 do not contribute to dental pulp sensitivity to cold

Cavβ surface charged residues contribute to the regulation of neuronal calcium channels

Heme molecule functions as an endogenous agonist of astrocyte TLR2 to contribute to secondary brain damage after intracerebral hemorrhage

Both local stability and dispersal contribute to metacommunity sensitivity to asynchronous habitat availability

MiR-136 modulates glioma cell sensitivity to temozolomide by targeting astrocyte elevated gene-1

Channels and Transporters in Astrocyte Volume Regulation in Health and Disease

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Imbalance and dysfunction of transient receptor potential channels contribute to the pathogenesis of hypertension

Specific ion channels contribute to key elements of pathology during secondary degeneration following neurotrauma

Overexpression of CPEB3 leads to astrocyte dysfunction

Astrocyte heterogeneity: from brain injury to repair

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