Correction: Network proteomics of the Lewy body dementia brain reveals presynaptic signatures distinct from Alzheimer’s disease

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Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: 1 Online-Ressource.

Language: Englisch

Bibliographic citation: Correction: Network proteomics of the Lewy body dementia brain reveals presynaptic signatures distinct from Alzheimer’s disease ; volume:19 ; number:1 ; day:11 ; month:10 ; year:2024 ; pages:1 ; date:12.2024
Molecular neurodegeneration ; 19, Heft 1 (11.10.2024), 1, 12.2024

Creator: Shantaraman, Anantharaman
Dammer, Eric B.
Ugochukwu, Obiadada
Duong, Duc M.
Yin, Luming
Carter, E. Kathleen
Gearing, Marla
Chen-Plotkin, Alice
Lee, Edward B.
Trojanowski, John Q.
Bennett, David A.
Lah, James J.
Levey, Allan I.
Seyfried, Nicholas T.
Higginbotham, Lenora

Contributor: SpringerLink (Online service)

DOI: 10.1186/s13024-024-00765-1

URN: urn:nbn:de:101:1-2412302105506.278043785977

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:37 AM CEST

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Shantaraman, Anantharaman
Dammer, Eric B.
Ugochukwu, Obiadada
Duong, Duc M.
Yin, Luming
Carter, E. Kathleen
Gearing, Marla
Chen-Plotkin, Alice
Lee, Edward B.
Trojanowski, John Q.
Bennett, David A.
Lah, James J.
Levey, Allan I.
Seyfried, Nicholas T.
Higginbotham, Lenora
SpringerLink (Online service)

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Correction: Network proteomics of the Lewy body dementia brain reveals presynaptic signatures distinct from Alzheimer’s disease

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Other Objects (12)

Network proteomics of the Lewy body dementia brain reveals presynaptic signatures distinct from Alzheimer’s disease

Plaque complement activation and cognitive loss in Alzheimer's disease

Single-nucleus RNA velocity reveals critical synaptic and cell-cycle dysregulations in neuropathologically confirmed Alzheimer’s disease

Genome-wide epistasis analysis for Alzheimer’s disease and implications for genetic risk prediction

Protein farnesylation is upregulated in Alzheimer’s human brains and neuron-specific suppression of farnesyltransferase mitigates pathogenic processes in Alzheimer’s model mice

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Epistatic effects of IGHG and FCGRIIB genes on the development of Alzheimer’s disease in African Americans

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The association of Lewy bodies with limbic-predominant age-related TDP-43 encephalopathy neuropathologic changes and their role in cognition and Alzheimer’s dementia in older persons

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Pathologic and clinical correlates of region-specific brain GFAP in Alzheimer’s disease

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Plaque complement activation and cognitive loss in Alzheimer's disease

Single-nucleus RNA velocity reveals critical synaptic and cell-cycle dysregulations in neuropathologically confirmed Alzheimer’s disease

Genome-wide epistasis analysis for Alzheimer’s disease and implications for genetic risk prediction

Protein farnesylation is upregulated in Alzheimer’s human brains and neuron-specific suppression of farnesyltransferase mitigates pathogenic processes in Alzheimer’s model mice

Specific serum antibody binding to phosphorylated and non-phosphorylated tau in non-cognitively impaired, mildly cognitively impaired, and Alzheimer’s disease subjects: an exploratory study

Epistatic effects of IGHG and FCGRIIB genes on the development of Alzheimer’s disease in African Americans

TDP-43 pathology in anterior temporal pole cortex in aging and Alzheimer’s disease

Mitigation of synaptic and memory impairments via F-actin stabilization in Alzheimer’s disease

The association of Lewy bodies with limbic-predominant age-related TDP-43 encephalopathy neuropathologic changes and their role in cognition and Alzheimer’s dementia in older persons

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Pathologic and clinical correlates of region-specific brain GFAP in Alzheimer’s disease

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Plaque complement activation and cognitive loss in Alzheimer's disease

Single-nucleus RNA velocity reveals critical synaptic and cell-cycle dysregulations in neuropathologically confirmed Alzheimer’s disease

Genome-wide epistasis analysis for Alzheimer’s disease and implications for genetic risk prediction

Protein farnesylation is upregulated in Alzheimer’s human brains and neuron-specific suppression of farnesyltransferase mitigates pathogenic processes in Alzheimer’s model mice

Specific serum antibody binding to phosphorylated and non-phosphorylated tau in non-cognitively impaired, mildly cognitively impaired, and Alzheimer’s disease subjects: an exploratory study

Epistatic effects of IGHG and FCGRIIB genes on the development of Alzheimer’s disease in African Americans

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