Hochschulschrift
S0X9 upstream regulation in human sex reversal and the requirement of Sox9, Sox8 and Dicer for adult mouse testis function and integrity
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Dimensions
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30 cm
- Extent
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XIX, 194 S.
- Language
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Englisch
- Notes
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Ill., graph. Darst.
Enth. außerdem Sonderabdr.
Freiburg (Breisgau), Univ., Diss., 2014 (Nur beschränkt für den Austausch)
- Classification
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Medizin, Gesundheit
Biowissenschaften, Biologie
- Creator
- Table of contents
- Rights
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Bei diesem Objekt liegt nur das Inhaltsverzeichnis digital vor. Der Zugriff darauf ist unbeschränkt möglich.
- Last update
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11.06.2025, 2:17 PM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Object type
- Hochschulschrift
Associated
Other Objects (12)
Sox9 and Sox8 protect the adult testis from male-to-female genetic reprogramming and complete degeneration
Estrogen suppresses SOX9 and activates markers of female development in a human testis-derived cell line
Human sex reversal is caused by duplication or deletion of core enhancers upstream of SOX9
Tool-based requirement traceability between requirement and design artifacts
SOX9-activated FARSA-AS1 predetermines cell growth, stemness, and metastasis in colorectal cancer through upregulating FARSA and SOX9
Differential transactivation of the upstream aggrecan enhancer regulated by PAX1/9 depends on SOX9-driven transactivation
A mutation creating an upstream initiation codon in the SOX9 5′ UTR causes acampomelic campomelic dysplasia
Mobile distributions and upstream challenges
GRUB upstream and distros cooperation
Sharing and Exploiting Requirement Decisions
SOX9 in organogenesis: shared and unique transcriptional functions
Intrafamilial Upstream Transfers and Household Production
Sox9 and Sox8 protect the adult testis from male-to-female genetic reprogramming and complete degeneration
Estrogen suppresses SOX9 and activates markers of female development in a human testis-derived cell line
Human sex reversal is caused by duplication or deletion of core enhancers upstream of SOX9
Tool-based requirement traceability between requirement and design artifacts
SOX9-activated FARSA-AS1 predetermines cell growth, stemness, and metastasis in colorectal cancer through upregulating FARSA and SOX9
Differential transactivation of the upstream aggrecan enhancer regulated by PAX1/9 depends on SOX9-driven transactivation
A mutation creating an upstream initiation codon in the SOX9 5′ UTR causes acampomelic campomelic dysplasia
Mobile distributions and upstream challenges
GRUB upstream and distros cooperation
Sharing and Exploiting Requirement Decisions
SOX9 in organogenesis: shared and unique transcriptional functions
Intrafamilial Upstream Transfers and Household Production
Sox9 and Sox8 protect the adult testis from male-to-female genetic reprogramming and complete degeneration
Estrogen suppresses SOX9 and activates markers of female development in a human testis-derived cell line
Human sex reversal is caused by duplication or deletion of core enhancers upstream of SOX9
Tool-based requirement traceability between requirement and design artifacts
SOX9-activated FARSA-AS1 predetermines cell growth, stemness, and metastasis in colorectal cancer through upregulating FARSA and SOX9
Differential transactivation of the upstream aggrecan enhancer regulated by PAX1/9 depends on SOX9-driven transactivation
A mutation creating an upstream initiation codon in the SOX9 5′ UTR causes acampomelic campomelic dysplasia
Mobile distributions and upstream challenges
GRUB upstream and distros cooperation
Sharing and Exploiting Requirement Decisions
SOX9 in organogenesis: shared and unique transcriptional functions