Copy-choice recombination during mitochondrial L-strand synthesis causes DNA deletions

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 2041-1723

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Copy-choice recombination during mitochondrial L-strand synthesis causes DNA deletions ; volume:10 ; number:1 ; day:15 ; month:2 ; year:2019 ; pages:1-10 ; date:12.2019
Nature Communications ; 10, Heft 1 (15.2.2019), 1-10, 12.2019

Classification: Naturwissenschaften

Creator: Persson, Örjan
Muthukumar, Yazh
Basu, Swaraj
Jenninger, Louise
Uhler, Jay P.
Berglund, Anna-Karin
McFarland, Robert
Taylor, Robert W.
Gustafsson, Claes M.
Larsson, Erik
Falkenberg, Maria

Contributor: SpringerLink (Online service)

DOI: 10.1038/s41467-019-08673-5

URN: urn:nbn:de:101:1-2019031321032693116691

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:53 AM CEST

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Associated

Persson, Örjan
Muthukumar, Yazh
Basu, Swaraj
Jenninger, Louise
Uhler, Jay P.
Berglund, Anna-Karin
McFarland, Robert
Taylor, Robert W.
Gustafsson, Claes M.
Larsson, Erik
Falkenberg, Maria
SpringerLink (Online service)

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Copy-choice recombination during mitochondrial L-strand synthesis causes DNA deletions

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Other Objects (12)

Comparing copy-number profiles under multi-copy amplifications and deletions

Analysis of mitochondrial DNA deletions in patients with ideopathic myositis

Analysis of meiotic recombination in 22q11.2, a region that frequently undergoes deletions and duplications

Mitochondrial genome deletions and minicircles are common in lice (Insecta: Phthiraptera)

Occurrence of large-scale mitochondrial DNA deletions in human colorectal cancer

Secondary structure of the human mitochondrial genome affects formation of deletions

Nuclear and mitochondrial genetic variants associated with mitochondrial DNA copy number

Large Common Mitochondrial DNA Deletions Are Associated with a Mitochondrial SNP T14798C Near the 3′ Breakpoints

Detection and quantification of mitochondrial DNA deletions from next-generation sequence data

Mitochondrial DNA copy number and incident atrial fibrillation

Copy number variations of the mitochondrial DNA as potential cause of mitochondrial diseases

Mice lacking the mitochondrial exonuclease MGME1 accumulate mtDNA deletions without developing progeria

Comparing copy-number profiles under multi-copy amplifications and deletions

Analysis of mitochondrial DNA deletions in patients with ideopathic myositis

Analysis of meiotic recombination in 22q11.2, a region that frequently undergoes deletions and duplications

Mitochondrial genome deletions and minicircles are common in lice (Insecta: Phthiraptera)

Occurrence of large-scale mitochondrial DNA deletions in human colorectal cancer

Secondary structure of the human mitochondrial genome affects formation of deletions

Nuclear and mitochondrial genetic variants associated with mitochondrial DNA copy number

Large Common Mitochondrial DNA Deletions Are Associated with a Mitochondrial SNP T14798C Near the 3′ Breakpoints

Detection and quantification of mitochondrial DNA deletions from next-generation sequence data

Mitochondrial DNA copy number and incident atrial fibrillation

Copy number variations of the mitochondrial DNA as potential cause of mitochondrial diseases

Mice lacking the mitochondrial exonuclease MGME1 accumulate mtDNA deletions without developing progeria

Comparing copy-number profiles under multi-copy amplifications and deletions

Analysis of mitochondrial DNA deletions in patients with ideopathic myositis

Analysis of meiotic recombination in 22q11.2, a region that frequently undergoes deletions and duplications

Mitochondrial genome deletions and minicircles are common in lice (Insecta: Phthiraptera)

Occurrence of large-scale mitochondrial DNA deletions in human colorectal cancer

Secondary structure of the human mitochondrial genome affects formation of deletions

Nuclear and mitochondrial genetic variants associated with mitochondrial DNA copy number

Large Common Mitochondrial DNA Deletions Are Associated with a Mitochondrial SNP T14798C Near the 3′ Breakpoints

Detection and quantification of mitochondrial DNA deletions from next-generation sequence data

Mitochondrial DNA copy number and incident atrial fibrillation

Copy number variations of the mitochondrial DNA as potential cause of mitochondrial diseases

Mice lacking the mitochondrial exonuclease MGME1 accumulate mtDNA deletions without developing progeria

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