Decreased transcription-coupled nucleotide excision repair capacity is associated with increased p53- and MLH1-independent apoptosis in response to cisplatin
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1471-2407
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Decreased transcription-coupled nucleotide excision repair capacity is associated with increased p53- and MLH1-independent apoptosis in response to cisplatin ; volume:10 ; number:1 ; day:14 ; month:5 ; year:2010 ; pages:1-10 ; date:12.2010
BMC cancer ; 10, Heft 1 (14.5.2010), 1-10, 12.2010
- Creator
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Stubbert, Lawton J.
- Contributor
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Smith, Jennifer M.
McKay, Bruce C.
SpringerLink (Online service)
- DOI
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10.1186/1471-2407-10-207
- URN
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urn:nbn:de:1111-2016030357342
- Rights
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Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:22 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Stubbert, Lawton J.
- Smith, Jennifer M.
- McKay, Bruce C.
- SpringerLink (Online service)
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MLH1 enhances the sensitivity of human endometrial carcinoma cells to cisplatin by activating the MLH1/c-Abl apoptosis signaling pathway
Structural basis of TFIIH activation for nucleotide excision repair
Structural and functional characterization of nucleotide excision repair proteins
Structure-function analysis of human nucleotide excision DNA repair
Decline of nucleotide excision repair capacity in aging Caenorhabditis elegans
Characterization of the multifunctional XPG protein during Nucleotide-excision-repair
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Nucleotide excision repair pathway modulating both cancer risk and therapy
Nucleotide Excision Repair : From Recognition to Incision of damaged DNA
Uncommon nucleotide excision repair phenotypes revealed by targeted high-throughput sequencing
Mechanism and regulation of DNA damage recognition in nucleotide excision repair
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MLH1 enhances the sensitivity of human endometrial carcinoma cells to cisplatin by activating the MLH1/c-Abl apoptosis signaling pathway
Structural basis of TFIIH activation for nucleotide excision repair
Structural and functional characterization of nucleotide excision repair proteins
Structure-function analysis of human nucleotide excision DNA repair
Decline of nucleotide excision repair capacity in aging Caenorhabditis elegans
Characterization of the multifunctional XPG protein during Nucleotide-excision-repair
Nucleotide excision repair deficiency in melanoma in response to UVA
Nucleotide excision repair pathway modulating both cancer risk and therapy
Nucleotide Excision Repair : From Recognition to Incision of damaged DNA
Uncommon nucleotide excision repair phenotypes revealed by targeted high-throughput sequencing
Mechanism and regulation of DNA damage recognition in nucleotide excision repair