Acyl-Ghrelin Attenuates Neurochemical and Motor Deficits in the 6-OHDA Model of Parkinson’s Disease
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1573-6830
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
Acyl-Ghrelin Attenuates Neurochemical and Motor Deficits in the 6-OHDA Model of Parkinson’s Disease ; day:15 ; month:9 ; year:2022 ; pages:1-8
Cellular and molecular neurobiology ; (15.9.2022), 1-8
- Urheber
-
Rees, Daniel
Beynon, Amy L.
Lelos, Mariah J.
Smith, Gaynor A.
Roberts, Luke D.
Phelps, Lyndsey
Dunnett, Stephen B.
Morgan, Alwena H.
Brown, Rowan M.
Wells, Timothy
Davies, Jeffrey S.
- Beteiligte Personen und Organisationen
-
SpringerLink (Online service)
- DOI
-
10.1007/s10571-022-01282-9
- URN
-
urn:nbn:de:101:1-2022120221325255305378
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
25.12.20252025, 15:29 MEZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Rees, Daniel
- Beynon, Amy L.
- Lelos, Mariah J.
- Smith, Gaynor A.
- Roberts, Luke D.
- Phelps, Lyndsey
- Dunnett, Stephen B.
- Morgan, Alwena H.
- Brown, Rowan M.
- Wells, Timothy
- Davies, Jeffrey S.
- SpringerLink (Online service)
Ähnliche Objekte (12)
Neurochemical changes in animal models of Parkinson’s disease
Imidazoleacetic Acid-Ribotide in the Rodent Striatum: A Putative Neurochemical Link Between Motor and Autonomic Deficits in Parkinson’s Disease
Modulating brain networks associated with cognitive deficits in Parkinson’s disease
Motor skill learning, retention, and control deficits in Parkinson´s disease
Quantitative assessment of gait and neurochemical correlation in a classical murine model of Parkinson’s disease
Continuous force measurements reveal no inhibitory control deficits in Parkinson’s disease
Pisa Syndrome in Parkinson’s Disease: Pathogenic Roles of Verticality Perception Deficits
β-Oxidation in ghrelin-producing cells is important for ghrelin acyl-modification
Disentangling perceptual judgment and online feedback deficits in Parkinson’s freezing of gait
Screening for cognitive deficits in Parkinson's disease with the Parkinson neuropsychometric dementia assessment (PANDA) instrument
Ghrelin Receptor Influence on Cocaine Reward is Not Directly Dependent on Peripheral Acyl-Ghrelin
Lenalidomide reduces microglial activation and behavioral deficits in a transgenic model of Parkinson’s disease
Neurochemical changes in animal models of Parkinson’s disease
Imidazoleacetic Acid-Ribotide in the Rodent Striatum: A Putative Neurochemical Link Between Motor and Autonomic Deficits in Parkinson’s Disease
Modulating brain networks associated with cognitive deficits in Parkinson’s disease
Motor skill learning, retention, and control deficits in Parkinson´s disease
Quantitative assessment of gait and neurochemical correlation in a classical murine model of Parkinson’s disease
Continuous force measurements reveal no inhibitory control deficits in Parkinson’s disease
Pisa Syndrome in Parkinson’s Disease: Pathogenic Roles of Verticality Perception Deficits
β-Oxidation in ghrelin-producing cells is important for ghrelin acyl-modification
Disentangling perceptual judgment and online feedback deficits in Parkinson’s freezing of gait
Screening for cognitive deficits in Parkinson's disease with the Parkinson neuropsychometric dementia assessment (PANDA) instrument
Ghrelin Receptor Influence on Cocaine Reward is Not Directly Dependent on Peripheral Acyl-Ghrelin
Lenalidomide reduces microglial activation and behavioral deficits in a transgenic model of Parkinson’s disease
Neurochemical changes in animal models of Parkinson’s disease
Imidazoleacetic Acid-Ribotide in the Rodent Striatum: A Putative Neurochemical Link Between Motor and Autonomic Deficits in Parkinson’s Disease
Modulating brain networks associated with cognitive deficits in Parkinson’s disease
Motor skill learning, retention, and control deficits in Parkinson´s disease
Quantitative assessment of gait and neurochemical correlation in a classical murine model of Parkinson’s disease
Continuous force measurements reveal no inhibitory control deficits in Parkinson’s disease
Pisa Syndrome in Parkinson’s Disease: Pathogenic Roles of Verticality Perception Deficits
β-Oxidation in ghrelin-producing cells is important for ghrelin acyl-modification
Disentangling perceptual judgment and online feedback deficits in Parkinson’s freezing of gait
Screening for cognitive deficits in Parkinson's disease with the Parkinson neuropsychometric dementia assessment (PANDA) instrument
Ghrelin Receptor Influence on Cocaine Reward is Not Directly Dependent on Peripheral Acyl-Ghrelin