Lamin A/C deficiency-mediated ROS elevation contributes to pathogenic phenotypes of dilated cardiomyopathy in iPSC model
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
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Englisch
- Bibliographic citation
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Lamin A/C deficiency-mediated ROS elevation contributes to pathogenic phenotypes of dilated cardiomyopathy in iPSC model ; volume:15 ; number:1 ; day:14 ; month:8 ; year:2024 ; pages:1-16 ; date:12.2024
Nature Communications ; 15, Heft 1 (14.8.2024), 1-16, 12.2024
- Creator
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Qiu, Hangyuan
Sun, Yaxun
Wang, Xiaochen
Gong, Tingyu
Su, Jun
Shen, Jiaxi
Zhou, Jingjun
Xia, Jiafeng
Wang, Hao
Meng, Xiangfu
Fu, Guosheng
Zhang, Donghui
Jiang, Chenyang
Liang, Ping
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s41467-024-51318-5
- URN
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urn:nbn:de:101:1-2411041118273.651052737113
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:25 AM CEST
Data provider
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Qiu, Hangyuan
- Sun, Yaxun
- Wang, Xiaochen
- Gong, Tingyu
- Su, Jun
- Shen, Jiaxi
- Zhou, Jingjun
- Xia, Jiafeng
- Wang, Hao
- Meng, Xiangfu
- Fu, Guosheng
- Zhang, Donghui
- Jiang, Chenyang
- Liang, Ping
- SpringerLink (Online service)
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Absence of cyclin-dependent kinase inhibitor p27 or p18 increases efficiency of iPSC generation without induction of iPSC genomic instability
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Suspension culture improves iPSC expansion and pluripotency phenotype
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Inborn errors of metabolism: Lessons from iPSC models
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