Presence of periodontitis may synergistically contribute to cancer progression via Treg and IL-6
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2045-2322
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Presence of periodontitis may synergistically contribute to cancer progression via Treg and IL-6 ; volume:12 ; number:1 ; day:8 ; month:7 ; year:2022 ; pages:1-9 ; date:12.2022
Scientific reports ; 12, Heft 1 (8.7.2022), 1-9, 12.2022
- Creator
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Kajihara, Ryo
Sakai, Hironori
Han, Yibing
Amari, Kei
Kawamoto, Makiko
Hakoyama, Yusuke
Nagashio, Sachiho
Yamada, Shin-ichi
Sanjo, Hideki
Kurita, Hiroshi
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s41598-022-15690-w
- URN
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urn:nbn:de:101:1-2022083019183839367097
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:34 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Kajihara, Ryo
- Sakai, Hironori
- Han, Yibing
- Amari, Kei
- Kawamoto, Makiko
- Hakoyama, Yusuke
- Nagashio, Sachiho
- Yamada, Shin-ichi
- Sanjo, Hideki
- Kurita, Hiroshi
- SpringerLink (Online service)
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Identification and characterization of causal genetic variants and non-coding RNAs that contribute to the risk of periodontitis
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Cholesterol suppresses human iTreg differentiation and nTreg function through mitochondria-related mechanisms
The interactive association of smoking and drinking levels with presence of periodontitis in South Korean adults
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Pathological conditions re-shape physiological Tregs into pathological Tregs
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Activated STAT3 signaling pathway by ligature-induced periodontitis could contribute to neuroinflammation and cognitive impairment in rats
Optimizing human Treg immunotherapy by Treg subset selection and E-selectin ligand expression
CD4+CCR8+ Tregs in ovarian cancer: a potential effector Tregs for immune regulation
Treg in CNS autoimmune disease
Identification and characterization of causal genetic variants and non-coding RNAs that contribute to the risk of periodontitis
Methanogenesis in the presence of oxygenic photosynthetic bacteria may contribute to global methane cycle
Cholesterol suppresses human iTreg differentiation and nTreg function through mitochondria-related mechanisms
The interactive association of smoking and drinking levels with presence of periodontitis in South Korean adults
Analysis of localized immune responses reveals presence of Th17 and Treg cells in cutaneous leishmaniasis due to Leishmania tropica
CTHRC1 + fibroblasts and SPP1 + macrophages synergistically contribute to pro-tumorigenic tumor microenvironment in pancreatic ductal adenocarcinoma
Pathological conditions re-shape physiological Tregs into pathological Tregs
Stuttgart Trauba-Treg
Activated STAT3 signaling pathway by ligature-induced periodontitis could contribute to neuroinflammation and cognitive impairment in rats
Optimizing human Treg immunotherapy by Treg subset selection and E-selectin ligand expression
CD4+CCR8+ Tregs in ovarian cancer: a potential effector Tregs for immune regulation
Treg in CNS autoimmune disease
Identification and characterization of causal genetic variants and non-coding RNAs that contribute to the risk of periodontitis
Methanogenesis in the presence of oxygenic photosynthetic bacteria may contribute to global methane cycle
Cholesterol suppresses human iTreg differentiation and nTreg function through mitochondria-related mechanisms
The interactive association of smoking and drinking levels with presence of periodontitis in South Korean adults