The PI3K inhibitor copanlisib synergizes with sorafenib to induce cell death in hepatocellular carcinoma
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2058-7716
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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The PI3K inhibitor copanlisib synergizes with sorafenib to induce cell death in hepatocellular carcinoma ; volume:5 ; number:1 ; day:5 ; month:4 ; year:2019 ; pages:1-12 ; date:12.2019
Cell death discovery ; 5, Heft 1 (5.4.2019), 1-12, 12.2019
- Creator
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s41420-019-0165-7
- URN
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urn:nbn:de:101:1-2019042520332226353763
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 11:00 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Ye, Liangtao
- Mayerle, Julia
- Ziesch, Andreas
- Reiter, Florian P.
- Gerbes, Alexander L.
- Toni, Enrico de
- SpringerLink (Online service)
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Exosomes derived from HCC cells induce sorafenib resistance in hepatocellular carcinoma both in vivo and in vitro
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Hydroxychloroquine synergizes with the PI3K inhibitor BKM120 to exhibit antitumor efficacy independent of autophagy
Novel FLT3/AURK multikinase inhibitor is efficacious against sorafenib-refractory and sorafenib-resistant hepatocellular carcinoma
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IGF-1R down regulates the sensitivity of hepatocellular carcinoma to sorafenib through the PI3K / akt and RAS / raf / ERK signaling pathways
Fe3+-binding transferrin nanovesicles encapsulating sorafenib induce ferroptosis in hepatocellular carcinoma
Cancer-associated fibroblasts induce sorafenib resistance of hepatocellular carcinoma cells through CXCL12/FOLR1
CD155 downregulation synergizes with adriamycin to induce breast cancer cell apoptosis
Exosomes derived from HCC cells induce sorafenib resistance in hepatocellular carcinoma both in vivo and in vitro
Unresectable Ectopic Hepatocellular Carcinoma Treated with Sorafenib
Niclosamide Exhibits Potent Anticancer Activity and Synergizes with Sorafenib in Human Renal Cell Cancer Cells
Topotecan synergizes with CHEK1 (CHK1) inhibitor to induce apoptosis in ovarian cancer cells
Melatonin Synergizes with Sorafenib to Suppress Pancreatic Cancer via Melatonin Receptor and PDGFR-β/STAT3 Pathway
Hydroxychloroquine synergizes with the PI3K inhibitor BKM120 to exhibit antitumor efficacy independent of autophagy
Novel FLT3/AURK multikinase inhibitor is efficacious against sorafenib-refractory and sorafenib-resistant hepatocellular carcinoma
A novel Mcl-1 inhibitor synergizes with venetoclax to induce apoptosis in cancer cells
IGF-1R down regulates the sensitivity of hepatocellular carcinoma to sorafenib through the PI3K / akt and RAS / raf / ERK signaling pathways
Fe3+-binding transferrin nanovesicles encapsulating sorafenib induce ferroptosis in hepatocellular carcinoma
Cancer-associated fibroblasts induce sorafenib resistance of hepatocellular carcinoma cells through CXCL12/FOLR1
CD155 downregulation synergizes with adriamycin to induce breast cancer cell apoptosis
Exosomes derived from HCC cells induce sorafenib resistance in hepatocellular carcinoma both in vivo and in vitro
Unresectable Ectopic Hepatocellular Carcinoma Treated with Sorafenib
Niclosamide Exhibits Potent Anticancer Activity and Synergizes with Sorafenib in Human Renal Cell Cancer Cells
Topotecan synergizes with CHEK1 (CHK1) inhibitor to induce apoptosis in ovarian cancer cells
Melatonin Synergizes with Sorafenib to Suppress Pancreatic Cancer via Melatonin Receptor and PDGFR-β/STAT3 Pathway
Hydroxychloroquine synergizes with the PI3K inhibitor BKM120 to exhibit antitumor efficacy independent of autophagy
Novel FLT3/AURK multikinase inhibitor is efficacious against sorafenib-refractory and sorafenib-resistant hepatocellular carcinoma
A novel Mcl-1 inhibitor synergizes with venetoclax to induce apoptosis in cancer cells