A threshold level of NFATc1 activity facilitates thymocyte differentiation and opposes notch-driven leukaemia development
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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Online-Ressource
- Language
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Englisch
- Bibliographic citation
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In: Nature Communications 2016, 7:11841. DOI: 10.1038/ncomms11841
- Event
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Veröffentlichung
- (where)
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Würzburg
- (who)
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Universität Würzburg
- (when)
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2021
- Creator
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Klein-Hessling, Stefan
Rudolf, Ronald
Muhammad, Khalid
Knobeloch, Klaus-Peter
Maqbool, Muhammad Ahmad
Cauchy, Pierre
Andrau, Jean-Christophe
Avots, Andris
Talora, Claudio
Ellenrieder, Volker
Screpanti, Isabella
Serfling, Edgar
Patra, Amiya Kumar
- DOI
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10.1038/ncomms11841
- URN
-
urn:nbn:de:bvb:20-opus-172974
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:20 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Klein-Hessling, Stefan
- Rudolf, Ronald
- Muhammad, Khalid
- Knobeloch, Klaus-Peter
- Maqbool, Muhammad Ahmad
- Cauchy, Pierre
- Andrau, Jean-Christophe
- Avots, Andris
- Talora, Claudio
- Ellenrieder, Volker
- Screpanti, Isabella
- Serfling, Edgar
- Patra, Amiya Kumar
- Universität Würzburg
Time of origin
- 2021
Other Objects (12)
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Presenilin-1 differentially facilitates endoproteolysis of the beta-amyloid precursor protein and Notch
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Sumoylation modulates NFATc1-mediated lymphokine gene expression
Extracellular C1qbp inhibits myogenesis by suppressing NFATc1
Notch Project
The TGFβ/Notch axis facilitates Müller cell-to-epithelial transition to ultimately form a chronic glial scar
NFATc1 as a therapeutic target in Burkitt’s lymphoma : = NFATc1 - ein Angriffspunkt zur Therapie des Burkitt-Lymphoms
Notch pathway connections in primary leukaemia samples of limited size
Identification of novel Notch target genes in T cell leukaemia
Physiological Notch Signaling Maintains Bone Homeostasis via RBPjk and Hey Upstream of NFATc1
Autoregulation of NFATc1 gene
Presenilin-1 differentially facilitates endoproteolysis of the beta-amyloid precursor protein and Notch
Hairy-cell leukaemia
Hairy cell leukaemia
Sumoylation modulates NFATc1-mediated lymphokine gene expression
Extracellular C1qbp inhibits myogenesis by suppressing NFATc1
Notch Project
The TGFβ/Notch axis facilitates Müller cell-to-epithelial transition to ultimately form a chronic glial scar
NFATc1 as a therapeutic target in Burkitt’s lymphoma : = NFATc1 - ein Angriffspunkt zur Therapie des Burkitt-Lymphoms
Notch pathway connections in primary leukaemia samples of limited size
Identification of novel Notch target genes in T cell leukaemia
Physiological Notch Signaling Maintains Bone Homeostasis via RBPjk and Hey Upstream of NFATc1
Autoregulation of NFATc1 gene
Presenilin-1 differentially facilitates endoproteolysis of the beta-amyloid precursor protein and Notch
Hairy-cell leukaemia
Hairy cell leukaemia
Sumoylation modulates NFATc1-mediated lymphokine gene expression
Extracellular C1qbp inhibits myogenesis by suppressing NFATc1
Notch Project
The TGFβ/Notch axis facilitates Müller cell-to-epithelial transition to ultimately form a chronic glial scar