TREM2 is associated with increased risk for Alzheimer’s disease in African Americans
- Standort
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1750-1326
- Umfang
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Online-Ressource
- Sprache
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Englisch
- Anmerkungen
-
online resource.
- Erschienen in
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TREM2 is associated with increased risk for Alzheimer’s disease in African Americans ; volume:10 ; number:1 ; day:10 ; month:4 ; year:2015 ; pages:1-7 ; date:12.2015
Molecular neurodegeneration ; 10, Heft 1 (10.4.2015), 1-7, 12.2015
- Urheber
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Jin, Sheng Chih
Carrasquillo, Minerva M.
Benitez, Bruno A.
Skorupa, Tara
Carrell, David
Patel, Dwani
Lincoln, Sarah
Krishnan, Siddharth
Kachadoorian, Michaela
Reitz, Christiane
Mayeux, Richard
Wingo, Thomas S.
Lah, James J.
Levey, Allan I.
Murrell, Jill
Hendrie, Hugh
Foroud, Tatiana
Graff-Radford, Neill R.
Goate, Alison M.
Cruchaga, Carlos
Ertekin-Taner, Nilüfer
- Beteiligte Personen und Organisationen
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SpringerLink (Online service)
- DOI
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10.1186/s13024-015-0016-9
- URN
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urn:nbn:de:101:1-2021080720531772509094
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
14.08.2025, 10:53 MESZ
Datenpartner
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Beteiligte
- Jin, Sheng Chih
- Carrasquillo, Minerva M.
- Benitez, Bruno A.
- Skorupa, Tara
- Carrell, David
- Patel, Dwani
- Lincoln, Sarah
- Krishnan, Siddharth
- Kachadoorian, Michaela
- Reitz, Christiane
- Mayeux, Richard
- Wingo, Thomas S.
- Lah, James J.
- Levey, Allan I.
- Murrell, Jill
- Hendrie, Hugh
- Foroud, Tatiana
- Graff-Radford, Neill R.
- Goate, Alison M.
- Cruchaga, Carlos
- Ertekin-Taner, Nilüfer
- SpringerLink (Online service)
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TREM2 upregulation correlates with 5-hydroxymethycytosine enrichment in Alzheimer’s disease hippocampus
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Rare TREM2 variants associated with Alzheimer’s disease display reduced cell surface expression
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TREM2 upregulation correlates with 5-hydroxymethycytosine enrichment in Alzheimer’s disease hippocampus
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Dynamic changes of CSF sTREM2 in preclinical Alzheimer’s disease: the CABLE study
The Trem2 R47H variant confers loss-of-function-like phenotypes in Alzheimer’s disease