CD300f:IL-5 cross-talk inhibits adipose tissue eosinophil homing and subsequent IL-4 production
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
2045-2322
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
CD300f:IL-5 cross-talk inhibits adipose tissue eosinophil homing and subsequent IL-4 production ; volume:7 ; number:1 ; day:19 ; month:7 ; year:2017 ; pages:1-15 ; date:12.2017
Scientific reports ; 7, Heft 1 (19.7.2017), 1-15, 12.2017
- Urheber
-
Rozenberg, Perri
- Beteiligte Personen und Organisationen
-
Reichman, Hadar
Zab-Bar, Israel
Itan, Michal
Pasmanik-Chor, Metsada
Bouffi, Carine
Qimron, Udi
Bachelet, Ido
Fulkerson, Patricia C.
Rothenberg, Marc E.
Munitz, Ariel
SpringerLink (Online service)
- DOI
-
10.1038/s41598-017-06397-4
- URN
-
urn:nbn:de:101:1-2018090510405127778406
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
15.08.2025, 07:31 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Rozenberg, Perri
- Reichman, Hadar
- Zab-Bar, Israel
- Itan, Michal
- Pasmanik-Chor, Metsada
- Bouffi, Carine
- Qimron, Udi
- Bachelet, Ido
- Fulkerson, Patricia C.
- Rothenberg, Marc E.
- Munitz, Ariel
- SpringerLink (Online service)
Ähnliche Objekte (12)
Evidence for eosinophil and IL-17 mediated inflammation in allergic rhinitis
IL-10+ regulatory B cells mitigate atopic dermatitis by suppressing eosinophil activation
Eosinophil‐derived IL ‐4 is necessary to establish the inflammatory structure in innate inflammation
Myeloid-IL4Rα is an indispensable link in IL-33-ILCs-IL-13-IL4Rα axis of eosinophil recruitment in murine lungs
Eosinophil Protein X Concentration Is Dependent on Eosinophil Concentration
ICON: Eosinophil Disorders
Eosinophil-independent IL-5 levels are increased in critically ill COVID-19 patients who survive
Homing. Song
Post-translational Tyrosine Nitration of Eosinophil Granule Toxins Mediated by Eosinophil Peroxidase
Hypoxia modulates human eosinophil function
Elucidating the crosstalk mechanism between IFN-gamma and IL-6 via mathematical modelling
Peripheral eosinophil count and eosinophil-to-lymphocyte ratio are associated with revision sinus surgery
Evidence for eosinophil and IL-17 mediated inflammation in allergic rhinitis
IL-10+ regulatory B cells mitigate atopic dermatitis by suppressing eosinophil activation
Eosinophil‐derived IL ‐4 is necessary to establish the inflammatory structure in innate inflammation
Myeloid-IL4Rα is an indispensable link in IL-33-ILCs-IL-13-IL4Rα axis of eosinophil recruitment in murine lungs
Eosinophil Protein X Concentration Is Dependent on Eosinophil Concentration
ICON: Eosinophil Disorders
Eosinophil-independent IL-5 levels are increased in critically ill COVID-19 patients who survive
Homing. Song
Post-translational Tyrosine Nitration of Eosinophil Granule Toxins Mediated by Eosinophil Peroxidase
Hypoxia modulates human eosinophil function
Elucidating the crosstalk mechanism between IFN-gamma and IL-6 via mathematical modelling
Peripheral eosinophil count and eosinophil-to-lymphocyte ratio are associated with revision sinus surgery
Evidence for eosinophil and IL-17 mediated inflammation in allergic rhinitis
IL-10+ regulatory B cells mitigate atopic dermatitis by suppressing eosinophil activation
Eosinophil‐derived IL ‐4 is necessary to establish the inflammatory structure in innate inflammation
Myeloid-IL4Rα is an indispensable link in IL-33-ILCs-IL-13-IL4Rα axis of eosinophil recruitment in murine lungs
Eosinophil Protein X Concentration Is Dependent on Eosinophil Concentration
ICON: Eosinophil Disorders
Eosinophil-independent IL-5 levels are increased in critically ill COVID-19 patients who survive
Homing. Song
Post-translational Tyrosine Nitration of Eosinophil Granule Toxins Mediated by Eosinophil Peroxidase
Hypoxia modulates human eosinophil function
Elucidating the crosstalk mechanism between IFN-gamma and IL-6 via mathematical modelling