- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
-
1 Online-Ressource.
- Sprache
-
Englisch
- Erschienen in
-
Gene duplication and deletion caused by over-replication at a fork barrier ; volume:14 ; number:1 ; day:25 ; month:11 ; year:2023 ; pages:1-15 ; date:12.2023
Nature Communications ; 14, Heft 1 (25.11.2023), 1-15, 12.2023
- Urheber
-
Oehler, Judith
Morrow, Carl A.
Whitby, Matthew C.
- Beteiligte Personen und Organisationen
-
SpringerLink (Online service)
- DOI
-
10.1038/s41467-023-43494-7
- URN
-
urn:nbn:de:101:1-2024021310431233838915
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
15.08.2025, 07:37 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Oehler, Judith
- Morrow, Carl A.
- Whitby, Matthew C.
- SpringerLink (Online service)
Ähnliche Objekte (12)
Replication termination without a replication fork trap
Human sex reversal is caused by duplication or deletion of core enhancers upstream of SOX9
Epilepsies in Children with 2q24.3 Deletion/Duplication
Prevention of unwanted recombination at damaged replication forks
Mechanism of chromosomal DNA replication initiation and replication fork stabilization in eukaryotes
The HLTF–PARP1 interaction in the progression and stability of damaged replication forks caused by methyl methanesulfonate
TFIP11 promotes replication fork reversal to preserve genome stability
RNAPII-dependent ATM signaling at collisions with replication forks
Using Gene Duplication and Gene Deletion in Evolution Strategies
Psychiatric disorders in children with 16p11.2 deletion and duplication
A model for DNA replication showing how dormant origins safeguard against replication fork failure
Budding yeast Rif1 binds to replication origins and protects DNA at blocked replication forks
Replication termination without a replication fork trap
Human sex reversal is caused by duplication or deletion of core enhancers upstream of SOX9
Epilepsies in Children with 2q24.3 Deletion/Duplication
Prevention of unwanted recombination at damaged replication forks
Mechanism of chromosomal DNA replication initiation and replication fork stabilization in eukaryotes
The HLTF–PARP1 interaction in the progression and stability of damaged replication forks caused by methyl methanesulfonate
TFIP11 promotes replication fork reversal to preserve genome stability
RNAPII-dependent ATM signaling at collisions with replication forks
Using Gene Duplication and Gene Deletion in Evolution Strategies
Psychiatric disorders in children with 16p11.2 deletion and duplication
A model for DNA replication showing how dormant origins safeguard against replication fork failure
Budding yeast Rif1 binds to replication origins and protects DNA at blocked replication forks
Replication termination without a replication fork trap
Human sex reversal is caused by duplication or deletion of core enhancers upstream of SOX9
Epilepsies in Children with 2q24.3 Deletion/Duplication
Prevention of unwanted recombination at damaged replication forks
Mechanism of chromosomal DNA replication initiation and replication fork stabilization in eukaryotes
The HLTF–PARP1 interaction in the progression and stability of damaged replication forks caused by methyl methanesulfonate
TFIP11 promotes replication fork reversal to preserve genome stability
RNAPII-dependent ATM signaling at collisions with replication forks
Using Gene Duplication and Gene Deletion in Evolution Strategies
Psychiatric disorders in children with 16p11.2 deletion and duplication
A model for DNA replication showing how dormant origins safeguard against replication fork failure