Self-administered version of the Fabry-associated pain questionnaire for adult patients
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource
- Language
-
Englisch
- Bibliographic citation
-
In: Orphanet Journal of Rare Diseases (2015) 10:113 DOI 10.1186/s13023-015-0325-7
- Event
-
Veröffentlichung
- (where)
-
Würzburg
- (who)
-
Universität Würzburg
- (when)
-
2017
- Creator
-
Magg, Barbara
Riegler, Christoph
Wiedmann, Silke
Heuschmann, Peter
Sommer, Claudia
Üçeyler, Nurcan
- URN
-
urn:nbn:de:bvb:20-opus-145294
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
25.03.2025, 1:49 PM CET
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Magg, Barbara
- Riegler, Christoph
- Wiedmann, Silke
- Heuschmann, Peter
- Sommer, Claudia
- Üçeyler, Nurcan
- Universität Würzburg
Time of origin
- 2017
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MDL-28170 Has No Analgesic Effect on CCI Induced Neuropathic Pain in Mice
The impact of inflammation, hypoxia, and vasculopathy on pain development in the α-galactosidase A mouse model of Morbus Fabry
Behandlungswechsel von Agalsidase beta zu Agalsidase alfa bei Morbus Fabry
Distinguishing phenotypes of Complex Regional Pain Syndrome
Local inflammatory mediators involved in neuropathic pain
The role of miR-21 in the pathophysiology of neuropathic pain using the model of B7-H1 knockout mice
English version of the self-administered Fabry Pain Questionnaire for adult patients
High-Dose Capsaicin for the Treatment of Neuropathic Pain: What We Know and What We Need to Know
MoKon - ein Ansatz zur wissensbasierten Konfiguration von Variantenerzeugnissen
Natural course of Guillain-Barré syndrome
The New Palace in Sans Souci Park
Tumor necrosis factor-α links heat and inflammation with Fabry pain
MDL-28170 Has No Analgesic Effect on CCI Induced Neuropathic Pain in Mice
The impact of inflammation, hypoxia, and vasculopathy on pain development in the α-galactosidase A mouse model of Morbus Fabry
Behandlungswechsel von Agalsidase beta zu Agalsidase alfa bei Morbus Fabry
Distinguishing phenotypes of Complex Regional Pain Syndrome
Local inflammatory mediators involved in neuropathic pain