- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource
- Language
-
Englisch
- Bibliographic citation
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Neuroinflammation and glial cell activation in pathogenesis of chronic pain ; volume:6 ; number:1 ; year:2015 ; pages:1-2 ; extent:2
Scandinavian journal of pain ; 6, Heft 1 (2015), 1-2 (gesamt 2)
- Creator
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Hansson, Elisabeth
- DOI
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10.1016/j.sjpain.2014.10.004
- URN
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urn:nbn:de:101:1-2024022713403371455415
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 11:01 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Hansson, Elisabeth
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Tauroursodeoxycholic acid reduces glial cell activation in an animal model of acute neuroinflammation
Calcineurin and glial signaling: neuroinflammation and beyond
Lipoxins A4 and B4 inhibit glial cell activation via CXCR3 signaling in acute retinal neuroinflammation
Chemokine CXCL13–CXCR5 signaling in neuroinflammation and pathogenesis of chronic pain and neurological diseases
Hyperammonemia induces glial activation, neuroinflammation and alters neurotransmitter receptors in hippocampus, impairing spatial learning: reversal by sulforaphane
Neuroinflammation induces glial aromatase expression in the uninjured songbird brain
A mouse model of hepatic encephalopathy: bile duct ligation induces brain ammonia overload, glial cell activation and neuroinflammation
Glial dysfunction and persistent neuropathic postsurgical pain
Etifoxine improves sensorimotor deficits and reduces glial activation, neuronal degeneration, and neuroinflammation in a rat model of traumatic brain injury
Activation of beta2-adrenergic receptors alleviates neuropathic pain hypersensitivity in mice: focus on spinal glial cells
Repeated electroacupuncture treatment attenuated hyperalgesia through suppression of spinal glial activation in chronic neuropathic pain rats
Perspectives in Pain Research 2014: Neuroinflammation and glial cell activation: The cause of transition from acute to chronic pain?
Tauroursodeoxycholic acid reduces glial cell activation in an animal model of acute neuroinflammation
Calcineurin and glial signaling: neuroinflammation and beyond
Lipoxins A4 and B4 inhibit glial cell activation via CXCR3 signaling in acute retinal neuroinflammation
Chemokine CXCL13–CXCR5 signaling in neuroinflammation and pathogenesis of chronic pain and neurological diseases
Hyperammonemia induces glial activation, neuroinflammation and alters neurotransmitter receptors in hippocampus, impairing spatial learning: reversal by sulforaphane
Neuroinflammation induces glial aromatase expression in the uninjured songbird brain
A mouse model of hepatic encephalopathy: bile duct ligation induces brain ammonia overload, glial cell activation and neuroinflammation
Glial dysfunction and persistent neuropathic postsurgical pain
Etifoxine improves sensorimotor deficits and reduces glial activation, neuronal degeneration, and neuroinflammation in a rat model of traumatic brain injury
Activation of beta2-adrenergic receptors alleviates neuropathic pain hypersensitivity in mice: focus on spinal glial cells