The RyR2-P2328S mutation downregulates Nav1.5 producing arrhythmic substrate in murine ventricles
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1432-2013
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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The RyR2-P2328S mutation downregulates Nav1.5 producing arrhythmic substrate in murine ventricles ; volume:468 ; number:4 ; day:6 ; month:11 ; year:2015 ; pages:655-665 ; date:4.2016
Pflügers Archiv ; 468, Heft 4 (6.11.2015), 655-665, 4.2016
- Creator
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Ning, Feifei
- Contributor
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Luo, Ling
Ahmad, Shiraz
Valli, Haseeb
Jeevaratnam, Kamalan
Wang, Tingzhong
Guzadhur, Laila
Yang, Dandan
Fraser, James A.
Huang, Christopher L.-H
Ma, Aiqun
Salvage, Samantha C.
SpringerLink (Online service)
- DOI
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10.1007/s00424-015-1750-0
- URN
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urn:nbn:de:1111-2016032635873
- Rights
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Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:59 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Ning, Feifei
- Luo, Ling
- Ahmad, Shiraz
- Valli, Haseeb
- Jeevaratnam, Kamalan
- Wang, Tingzhong
- Guzadhur, Laila
- Yang, Dandan
- Fraser, James A.
- Huang, Christopher L.-H
- Ma, Aiqun
- Salvage, Samantha C.
- SpringerLink (Online service)
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Ryanodine Receptor 2 (RyR2) underlies maintenance and remodeling of dendritic spines
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“Ryanopathies” and RyR2 dysfunctions: can we further decipher them using in vitro human disease models?
Promiscuous attraction of ligands within the ATP binding site of RyR2 promotes diverse gating behaviour
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Fushenmu treatment ameliorates RyR2 with related metabolites in a zebrafish model of barium chloride induced arrhythmia
Konditionelle Gendeletion des Ryanodinrezeptors RyR2 im Herzen der Maus
Konditionelle Gendeletion des Ryanodinrezeptors RyR2 im Herzen der Maus
Increased RyR2 activity is exacerbated by calcium leak-induced mitochondrial ROS
Ryanodine Receptor 2 (RyR2) underlies maintenance and remodeling of dendritic spines
Funktionelle Konsequenzen einer Einzelnukleotid-Polymorphismus-induzierten Mutation im humanen kardialen Ryanodinrezeptor (RyR2)
Direct regulation of the cardiac ryanodine receptor (RyR2) by O-GlcNAcylation
R2, 7 R2, 8 R2, 9
R2, 4 R2, 5 R2, 6
“Ryanopathies” and RyR2 dysfunctions: can we further decipher them using in vitro human disease models?
Promiscuous attraction of ligands within the ATP binding site of RyR2 promotes diverse gating behaviour
Ry
Fushenmu treatment ameliorates RyR2 with related metabolites in a zebrafish model of barium chloride induced arrhythmia
Konditionelle Gendeletion des Ryanodinrezeptors RyR2 im Herzen der Maus
Konditionelle Gendeletion des Ryanodinrezeptors RyR2 im Herzen der Maus
Increased RyR2 activity is exacerbated by calcium leak-induced mitochondrial ROS
Ryanodine Receptor 2 (RyR2) underlies maintenance and remodeling of dendritic spines
Funktionelle Konsequenzen einer Einzelnukleotid-Polymorphismus-induzierten Mutation im humanen kardialen Ryanodinrezeptor (RyR2)
Direct regulation of the cardiac ryanodine receptor (RyR2) by O-GlcNAcylation
R2, 7 R2, 8 R2, 9
R2, 4 R2, 5 R2, 6
“Ryanopathies” and RyR2 dysfunctions: can we further decipher them using in vitro human disease models?
Promiscuous attraction of ligands within the ATP binding site of RyR2 promotes diverse gating behaviour
Ry