Local leptin production in osteoarthritis subchondral osteoblasts may be responsible for their abnormal phenotypic expression
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1478-6354
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Local leptin production in osteoarthritis subchondral osteoblasts may be responsible for their abnormal phenotypic expression ; volume:12 ; number:1 ; day:8 ; month:2 ; year:2010 ; pages:1-13 ; date:2.2010
Arthritis Research & Therapy ; 12, Heft 1 (8.2.2010), 1-13, 2.2010
- Creator
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Mutabaruka, Marie-Solange
- Contributor
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Aoulad Aissa, Mohamed
Delalandre, Aline
Lavigne, Martin
Lajeunesse, Daniel
SpringerLink (Online service)
- DOI
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10.1186/ar2925
- URN
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urn:nbn:de:1111-2016050314281
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:55 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Mutabaruka, Marie-Solange
- Aoulad Aissa, Mohamed
- Delalandre, Aline
- Lavigne, Martin
- Lajeunesse, Daniel
- SpringerLink (Online service)
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Abnormal insulin-like growth factor 1 signaling in human osteoarthritic subchondral bone osteoblasts
Hypoxia and vitamin D differently contribute to leptin and dickkopf-related protein 2 production in human osteoarthritic subchondral bone osteoblasts
Relationship between abnormal osteoblasts and cellular immunity in multiple myeloma
Osteoclasts secrete leukemia inhibitory factor to promote abnormal bone remodeling of subchondral bone in osteoarthritis
Endothelial PDGF-BB/PDGFR-β signaling promotes osteoarthritis by enhancing angiogenesis-dependent abnormal subchondral bone formation
Wnt16 Overexpression in Osteoblasts Increases the Subchondral Bone Mass but has no Impact on Osteoarthritis in Young Adult Female Mice
Chondroitin and glucosamine sulfate in combination decrease the pro-resorptive properties of human osteoarthritis subchondral bone osteoblasts: a basic science study
MicroRNA-29a in Osteoblasts Represses High-Fat Diet-Mediated Osteoporosis and Body Adiposis through Targeting Leptin
Leptin signaling and leptin resistance
Knee osteoarthritis patients with more subchondral cysts have altered tibial subchondral bone mineral density
Leptin
Subchondral insufficiency fractures, subchondral insufficiency fractures with osteonecrosis, and other apparently spontaneous subchondral bone lesions of the knee—pathogenesis and diagnosis at imaging
Abnormal insulin-like growth factor 1 signaling in human osteoarthritic subchondral bone osteoblasts
Hypoxia and vitamin D differently contribute to leptin and dickkopf-related protein 2 production in human osteoarthritic subchondral bone osteoblasts
Relationship between abnormal osteoblasts and cellular immunity in multiple myeloma
Osteoclasts secrete leukemia inhibitory factor to promote abnormal bone remodeling of subchondral bone in osteoarthritis
Endothelial PDGF-BB/PDGFR-β signaling promotes osteoarthritis by enhancing angiogenesis-dependent abnormal subchondral bone formation
Wnt16 Overexpression in Osteoblasts Increases the Subchondral Bone Mass but has no Impact on Osteoarthritis in Young Adult Female Mice
Chondroitin and glucosamine sulfate in combination decrease the pro-resorptive properties of human osteoarthritis subchondral bone osteoblasts: a basic science study
MicroRNA-29a in Osteoblasts Represses High-Fat Diet-Mediated Osteoporosis and Body Adiposis through Targeting Leptin
Leptin signaling and leptin resistance
Knee osteoarthritis patients with more subchondral cysts have altered tibial subchondral bone mineral density
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