Osteopontin Regulates Endometrial Stromal Cell Migration in Endometriosis through the PI3K Pathway : Osteopontin Regulates Endometrial Cell Migration in Endometriosis
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1933-7205
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Osteopontin Regulates Endometrial Stromal Cell Migration in Endometriosis through the PI3K Pathway ; day:9 ; month:9 ; year:2020 ; pages:1-12
Reproductive sciences ; (9.9.2020), 1-12
- Creator
-
Fu, Xiaoxia
Yao, Mengyun
Ye, Chaoshuang
Fang, Tao
Wu, Ruijin
- Contributor
-
SpringerLink (Online service)
- DOI
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10.1007/s43032-020-00301-8
- URN
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urn:nbn:de:101:1-2020102921454431211256
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:23 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Fu, Xiaoxia
- Yao, Mengyun
- Ye, Chaoshuang
- Fang, Tao
- Wu, Ruijin
- SpringerLink (Online service)
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Endometriosis
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Altered differentiation of endometrial mesenchymal stromal fibroblasts is associated with endometriosis susceptibility
Single-cell analysis of menstrual endometrial tissues defines phenotypes associated with endometriosis
Increased α2-6 sialylation of endometrial cells contributes to the development of endometriosis
Endometrial stem/progenitor cells and their roles in immunity, clinical application, and endometriosis
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Endometriosis
Osteopontin regulates proliferation, apoptosis, and migration of murine claudin-low mammary tumor cells
BIBR1532 Affects Endometrial Cell Proliferation, Migration, and Invasion in Endometriosis via Telomerase Inhibition and MAPK Signaling
The influence of menstrual cycle and endometriosis on endometrial methylome
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MiR-449a regulates caprine endometrial stromal cell apoptosis and endometrial receptivity
Alcohol exposure decreases osteopontin expression during fracture healing and osteopontin-mediated mesenchymal stem cell migration in vitro
Altered differentiation of endometrial mesenchymal stromal fibroblasts is associated with endometriosis susceptibility
Single-cell analysis of menstrual endometrial tissues defines phenotypes associated with endometriosis
Increased α2-6 sialylation of endometrial cells contributes to the development of endometriosis
Endometrial stem/progenitor cells and their roles in immunity, clinical application, and endometriosis
Plasma High Mobility Group Box 1 (HMGB1), Osteopontin (OPN), and Hyaluronic Acid (HA) as Admissible Biomarkers for Endometriosis