The new IL-1 family member IL-1F8 stimulates production of inflammatory mediators by synovial fibroblasts and articular chondrocytes
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1478-6354
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
-
The new IL-1 family member IL-1F8 stimulates production of inflammatory mediators by synovial fibroblasts and articular chondrocytes ; volume:8 ; number:3 ; day:28 ; month:4 ; year:2006 ; pages:1-11 ; date:6.2006
Arthritis Research & Therapy ; 8, Heft 3 (28.4.2006), 1-11, 6.2006
- Creator
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Magne, David
- Contributor
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Palmer, Gaby
Barton, Jenny L.
Mezin, Françoise
Talabot-Ayer, Dominique
Bas, Sylvette
Duffy, Trevor
Noger, Marcus
Guerne, Pierre-Andre
Nicklin, Martin JH
Gabay, Cem
SpringerLink (Online service)
- DOI
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10.1186/ar1946
- URN
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urn:nbn:de:1111-201605034469
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:49 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Magne, David
- Palmer, Gaby
- Barton, Jenny L.
- Mezin, Françoise
- Talabot-Ayer, Dominique
- Bas, Sylvette
- Duffy, Trevor
- Noger, Marcus
- Guerne, Pierre-Andre
- Nicklin, Martin JH
- Gabay, Cem
- SpringerLink (Online service)
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Exosomes from IL-1β stimulated synovial fibroblasts induce osteoarthritic changes in articular chondrocytes
Dexamethasone stimulates expression of C-type Natriuretic Peptide in chondrocytes
The role of synovial fluid cytokines IL-6, IL-23 and IL-17 in the pathogenesis and persistence of synovial inflammation in JIA patients
The active metabolite of leflunomide, A77 1726, increases the production of IL-1 receptor antagonist in human synovial fibroblasts and articular chondrocytes
Norepinephrine modulates IL-1β-induced catabolic response of human chondrocytes
Nurr1 dependent regulation of pro-inflammatory mediators in immortalised synovial fibroblasts
Extracellular vesicles from platelet-poor plasma possess anti-inflammatory and anti-catabolic effects in chondrocytes stimulated with IL-1β or synovial membrane-conditioned media
IL-40 is up-regulated in the synovial fluid and cartilage of osteoarthritis patients and contributes to the alteration of chondrocytes phenotype in vitro
Airway allergen exposure stimulates bone marrow eosinophilia partly via IL-9
Exogenous glucosamine globally protects chondrocytes from the arthritogenic effects of IL-1β
miR-21-5p protects IL-1β-induced human chondrocytes from degradation
IL-1β Regulates FHL2 and Other Cytoskeleton-Related Genes in Human Chondrocytes
Exosomes from IL-1β stimulated synovial fibroblasts induce osteoarthritic changes in articular chondrocytes
Dexamethasone stimulates expression of C-type Natriuretic Peptide in chondrocytes
The role of synovial fluid cytokines IL-6, IL-23 and IL-17 in the pathogenesis and persistence of synovial inflammation in JIA patients
The active metabolite of leflunomide, A77 1726, increases the production of IL-1 receptor antagonist in human synovial fibroblasts and articular chondrocytes
Norepinephrine modulates IL-1β-induced catabolic response of human chondrocytes
Nurr1 dependent regulation of pro-inflammatory mediators in immortalised synovial fibroblasts
Extracellular vesicles from platelet-poor plasma possess anti-inflammatory and anti-catabolic effects in chondrocytes stimulated with IL-1β or synovial membrane-conditioned media
IL-40 is up-regulated in the synovial fluid and cartilage of osteoarthritis patients and contributes to the alteration of chondrocytes phenotype in vitro
Airway allergen exposure stimulates bone marrow eosinophilia partly via IL-9
Exogenous glucosamine globally protects chondrocytes from the arthritogenic effects of IL-1β
miR-21-5p protects IL-1β-induced human chondrocytes from degradation