The nucleolar remodeling complex NoRC mediates heterochromatin formation and silencing of ribosomal gene transcription
- Location
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München, Bayerische Staatsbibliothek -- 4 Z 92.354-32#S.393-396
- Extent
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1 Online-Ressource (S. 393 - 396)
- Language
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Englisch
- Notes
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Ill.
In: Nature genetics, Vol. 32, No. 3, 2002. - New York, NY : Nature America
- Event
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Veröffentlichung
- (where)
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New York, NY
- (who)
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Nature America
- (when)
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2002
- Creator
- URN
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urn:nbn:de:bvb:12-bsb00065157-8
- Last update
-
16.04.2025, 8:48 AM CEST
Data provider
This object is provided by:
Bayerische Staatsbibliothek. If you have any questions about the object, please contact the data provider.
Bayerische Staatsbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Santoro, Raffaella
- Li, Junwei
- Grummt, Ingrid
- Nature America
Time of origin
- 2002
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Relevance of heterochromatin topology in genome silencing during (retro) differentiation
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An inventory of yeast proteins associated with nucleolar and ribosomal components
Epigeneitc silencing of ribosomal RNA genes by Mybbp1a
The molecular basis for stability of heterochromatin-mediated silencing in mammals
Histone chaperone Spt16p is required for heterochromatin mediated silencing in budding yeast
Silencing and activating heterochromatin
Silencing and activating heterochromatin
NoRC, a novel chromatin remodeling complex involved in ribosomal RNA gene silencing
NoRC, a novel chromatin remodeling complex involved in ribosomal RNA gene silencing
Mercury chloride alters heterochromatin domain organization and nucleolar activity in mouse liver
Nucleolar and ribosomal dysfunction—a common pathomechanism in childhood progerias?
Relevance of heterochromatin topology in genome silencing during (retro) differentiation
Relevance of heterochromatin topology in genome silencing during (retro) differentiation
An inventory of yeast proteins associated with nucleolar and ribosomal components
Epigeneitc silencing of ribosomal RNA genes by Mybbp1a
The molecular basis for stability of heterochromatin-mediated silencing in mammals