Hochschulschrift
Interactions between the mycotoxin deoxynivalenol and lipopolysaccharides on the protein metabolism and the immune system of pigs
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
Hannover, Tierärztliche Hochsch., Diss., 2014
- Klassifikation
-
Landwirtschaft, Veterinärmedizin
- Ereignis
-
Veröffentlichung
- (wo)
-
Hannover
- (wer)
-
Bibliothek der Tierärztlichen Hochschule Hannover
- (wann)
-
2014
- Urheber
-
Kullik, Kristin
- URN
-
urn:nbn:de:gbv:95-105718
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
15.01.2025, 14:21 MEZ
Datenpartner
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Objekttyp
- Hochschulschrift
Beteiligte
- Kullik, Kristin
- Bibliothek der Tierärztlichen Hochschule Hannover
Entstanden
- 2014
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On the metabolism and toxicokinetics of deoxynivalenol and zearalenone in endotoxaemic pigs
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Humans significantly metabolize and excrete the mycotoxin deoxynivalenol and its modified form deoxynivalenol-3-glucoside within 24 hours
The mycotoxin deoxynivalenol activates GABAergic neurons in the reward system and inhibits feeding and maternal behaviours
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Structure–function characterization of an aldo–keto reductase involved in detoxification of the mycotoxin, deoxynivalenol
Does chronical deoxynivalenol-feeding modulate the immune response in endotoxaemic pigs?
Clinical impact of deoxynivalenol, 3-acetyl-deoxynivalenol and 15-acetyl-deoxynivalenol on the severity of an experimental Mycoplasma hyopneumoniae infection in pigs
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On the metabolism and toxicokinetics of deoxynivalenol and zearalenone in endotoxaemic pigs
The transcription factor FgSfp1 orchestrates mycotoxin deoxynivalenol biosynthesis in Fusarium graminearum
Humans significantly metabolize and excrete the mycotoxin deoxynivalenol and its modified form deoxynivalenol-3-glucoside within 24 hours
The mycotoxin deoxynivalenol activates GABAergic neurons in the reward system and inhibits feeding and maternal behaviours
Cryphonectria hypovirus 1 infection suppresses the pathogenicity but increases the mycotoxin deoxynivalenol production of Fusarium graminearum
Exposure to the mycotoxin deoxynivalenol reduces the transport of conjugated bile acids by intestinal Caco-2 cells
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