- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1478-6354
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Angiotensin II induces skin fibrosis: a novel mouse model of dermal fibrosis ; volume:14 ; number:4 ; day:22 ; month:8 ; year:2012 ; pages:1-12 ; date:8.2012
Arthritis Research & Therapy ; 14, Heft 4 (22.8.2012), 1-12, 8.2012
- Creator
-
Stawski, Lukasz
- Contributor
-
Han, Rong
Bujor, Andreea M.
Trojanowska, Maria
SpringerLink (Online service)
- DOI
-
10.1186/ar4028
- URN
-
urn:nbn:de:1111-2016050323251
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:44 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Stawski, Lukasz
- Han, Rong
- Bujor, Andreea M.
- Trojanowska, Maria
- SpringerLink (Online service)
Other Objects (12)
Fat transplantation induces dermal adipose regeneration and reverses skin fibrosis through dedifferentiation and redifferentiation of adipocytes
Oxidative Stress and Skin Fibrosis
Reconstruction of dermal layers evaluated by high-frequency ultrasound following treatment for skin fibrosis
Activation of pregnane X receptor inhibits experimental dermal fibrosis
Elucidating the cellular mechanism for E2-induced dermal fibrosis
Blockade of canonical Wnt signalling ameliorates experimental dermal fibrosis
Stimulation of soluble guanylate cyclase reduces experimental dermal fibrosis
Inhibition of glycogen synthase kinase 3β induces dermal fibrosis by activation of the canonical Wnt pathway
Inhibition of hedgehog signalling prevents experimental fibrosis and induces regression of established fibrosis
Antibody therapy can enhance AngiotensinII-induced myocardial fibrosis
![Inhibition of glycogen-synthase-kinase 3β [3-beta] induces dermal fibrosis by activation of the canonical Wnt pathway](/assets/placeholder/searchResultMediaNoDigitisedMedia.png)
Inhibition of glycogen-synthase-kinase 3β [3-beta] induces dermal fibrosis by activation of the canonical Wnt pathway
Advances of Nanobiomaterials for Treating Skin Pathological Fibrosis
Fat transplantation induces dermal adipose regeneration and reverses skin fibrosis through dedifferentiation and redifferentiation of adipocytes
Oxidative Stress and Skin Fibrosis
Reconstruction of dermal layers evaluated by high-frequency ultrasound following treatment for skin fibrosis
Activation of pregnane X receptor inhibits experimental dermal fibrosis
Elucidating the cellular mechanism for E2-induced dermal fibrosis
Blockade of canonical Wnt signalling ameliorates experimental dermal fibrosis
Stimulation of soluble guanylate cyclase reduces experimental dermal fibrosis
Inhibition of glycogen synthase kinase 3β induces dermal fibrosis by activation of the canonical Wnt pathway
Inhibition of hedgehog signalling prevents experimental fibrosis and induces regression of established fibrosis
Antibody therapy can enhance AngiotensinII-induced myocardial fibrosis
Inhibition of glycogen-synthase-kinase 3β [3-beta] induces dermal fibrosis by activation of the canonical Wnt pathway
Advances of Nanobiomaterials for Treating Skin Pathological Fibrosis
Fat transplantation induces dermal adipose regeneration and reverses skin fibrosis through dedifferentiation and redifferentiation of adipocytes
Oxidative Stress and Skin Fibrosis
Reconstruction of dermal layers evaluated by high-frequency ultrasound following treatment for skin fibrosis
Activation of pregnane X receptor inhibits experimental dermal fibrosis
Elucidating the cellular mechanism for E2-induced dermal fibrosis
Blockade of canonical Wnt signalling ameliorates experimental dermal fibrosis
Stimulation of soluble guanylate cyclase reduces experimental dermal fibrosis
Inhibition of glycogen synthase kinase 3β induces dermal fibrosis by activation of the canonical Wnt pathway
Inhibition of hedgehog signalling prevents experimental fibrosis and induces regression of established fibrosis
Antibody therapy can enhance AngiotensinII-induced myocardial fibrosis
Inhibition of glycogen-synthase-kinase 3β [3-beta] induces dermal fibrosis by activation of the canonical Wnt pathway