- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1750-1326
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Remodeling Alzheimer-amyloidosis models by seeding ; volume:16 ; number:1 ; day:15 ; month:2 ; year:2021 ; pages:1-11 ; date:12.2021
Molecular neurodegeneration ; 16, Heft 1 (15.2.2021), 1-11, 12.2021
- Creator
-
Ulm, Brittany S.
Borchelt, David R.
Moore, Brenda D.
- Contributor
-
SpringerLink (Online service)
- DOI
-
10.1186/s13024-021-00429-4
- URN
-
urn:nbn:de:101:1-2021040500404113570781
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:58 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Ulm, Brittany S.
- Borchelt, David R.
- Moore, Brenda D.
- SpringerLink (Online service)
Other Objects (12)
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Beyond genetics: including the environmental dimension in amyloidosis mouse models for Alzheimer’s disease
A[beta] seeding as a tool to study cerebral amyloidosis and associated pathology
Amyloid seeding as a disease mechanism and treatment target in transthyretin cardiac amyloidosis
Cathepsin B Improves ß-Amyloidosis and Learning and Memory in Models of Alzheimer’s Disease
Anatomic survey of seeding in Alzheimer’s disease brains reveals unexpected patterns
Ultrasensitive tau biosensor cells detect no seeding in Alzheimer’s disease CSF
Amyloid pathology arrangements in Alzheimer’s disease brains modulate in vivo seeding capability
Sex-specific hypothalamic neuropathology and glucose metabolism in an amyloidosis transgenic mouse model of Alzheimer’s disease
Insulin deficiency exacerbates cerebral amyloidosis and behavioral deficits in an Alzheimer transgenic mouse model
Correction to: Anatomic survey of seeding in Alzheimer’s disease brains reveals unexpected patterns
Effects of SPI1-mediated transcriptome remodeling on Alzheimer’s disease-related phenotypes in mouse models of Aβ amyloidosis
Microglial CD2AP deficiency exerts protection in an Alzheimer’s disease model of amyloidosis
Beyond genetics: including the environmental dimension in amyloidosis mouse models for Alzheimer’s disease
A[beta] seeding as a tool to study cerebral amyloidosis and associated pathology
Amyloid seeding as a disease mechanism and treatment target in transthyretin cardiac amyloidosis
Cathepsin B Improves ß-Amyloidosis and Learning and Memory in Models of Alzheimer’s Disease
Anatomic survey of seeding in Alzheimer’s disease brains reveals unexpected patterns
Ultrasensitive tau biosensor cells detect no seeding in Alzheimer’s disease CSF
Amyloid pathology arrangements in Alzheimer’s disease brains modulate in vivo seeding capability
Sex-specific hypothalamic neuropathology and glucose metabolism in an amyloidosis transgenic mouse model of Alzheimer’s disease
Insulin deficiency exacerbates cerebral amyloidosis and behavioral deficits in an Alzheimer transgenic mouse model
Correction to: Anatomic survey of seeding in Alzheimer’s disease brains reveals unexpected patterns
Effects of SPI1-mediated transcriptome remodeling on Alzheimer’s disease-related phenotypes in mouse models of Aβ amyloidosis
Microglial CD2AP deficiency exerts protection in an Alzheimer’s disease model of amyloidosis
Beyond genetics: including the environmental dimension in amyloidosis mouse models for Alzheimer’s disease
A[beta] seeding as a tool to study cerebral amyloidosis and associated pathology
Amyloid seeding as a disease mechanism and treatment target in transthyretin cardiac amyloidosis
Cathepsin B Improves ß-Amyloidosis and Learning and Memory in Models of Alzheimer’s Disease
Anatomic survey of seeding in Alzheimer’s disease brains reveals unexpected patterns
Ultrasensitive tau biosensor cells detect no seeding in Alzheimer’s disease CSF
Amyloid pathology arrangements in Alzheimer’s disease brains modulate in vivo seeding capability
Sex-specific hypothalamic neuropathology and glucose metabolism in an amyloidosis transgenic mouse model of Alzheimer’s disease
Insulin deficiency exacerbates cerebral amyloidosis and behavioral deficits in an Alzheimer transgenic mouse model