Association of HMGB1 polymorphisms with outcome in patients with systemic inflammatory response syndrome
- Standort
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1364-8535
- Umfang
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Online-Ressource
- Sprache
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Englisch
- Anmerkungen
-
online resource.
- Erschienen in
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Association of HMGB1 polymorphisms with outcome in patients with systemic inflammatory response syndrome ; volume:12 ; number:3 ; day:24 ; month:06 ; year:2008 ; pages:1-10 ; date:6.2008
Critical care ; 12, Heft 3 (24.06.2008), 1-10, 6.2008
- Urheber
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Kornblit, Brian
- Beteiligte Personen und Organisationen
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Munthe-Fog, Lea
Madsen, Hans O.
Strøm, Jens
Vindeløv, Lars
Garred, Peter
SpringerLink (Online service)
- DOI
-
10.1186/cc6935
- URN
-
urn:nbn:de:1111-2016080819927
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
14.08.2025, 10:50 MESZ
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Beteiligte
- Kornblit, Brian
- Munthe-Fog, Lea
- Madsen, Hans O.
- Strøm, Jens
- Vindeløv, Lars
- Garred, Peter
- SpringerLink (Online service)
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Systemic inflammatory markers in patients with polyneuropathies
Increased levels of HMGB1 and pro-inflammatory cytokines in children with febrile seizures
Therapeutic administration of etoposide coincides with reduced systemic HMGB1 levels in macrophage activation syndrome
Urine levels of HMGB1 in Systemic Lupus Erythematosus patients with and without renal manifestations
Genetic polymorphisms associated with the inflammatory response in bacterial meningitis
Association of inflammatory gene polymorphisms with mechanical heart valve reoperation
Vesicular HMGB1 release from neurons stressed with spreading depolarization enables confined inflammatory signaling to astrocytes
The acidic intrinsically disordered region of the inflammatory mediator HMGB1 mediates fuzzy interactions with CXCL12
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Alarmin HMGB1 induces systemic and brain inflammatory exacerbation in post-stroke infection rat model
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Increased levels of HMGB1 and pro-inflammatory cytokines in children with febrile seizures
Therapeutic administration of etoposide coincides with reduced systemic HMGB1 levels in macrophage activation syndrome
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