Oocytes maintain ROS-free mitochondrial metabolism by suppressing complex I

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1476-4687

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Oocytes maintain ROS-free mitochondrial metabolism by suppressing complex I ; volume:607 ; number:7920 ; day:20 ; month:7 ; year:2022 ; pages:756-761 ; date:7.2022
Nature ; 607, Heft 7920 (20.7.2022), 756-761, 7.2022

Creator: Rodríguez-Nuevo, Aida
Torres-Sanchez, Ariadna
Duran, Juan M.
Guirior, Cristian De
Martínez-Zamora, Maria Angeles
Böke, Elvan

Contributor: SpringerLink (Online service)

DOI: 10.1038/s41586-022-04979-5

URN: urn:nbn:de:101:1-2022101514123047919395

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:22 AM CEST

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Associated

Rodríguez-Nuevo, Aida
Torres-Sanchez, Ariadna
Duran, Juan M.
Guirior, Cristian De
Martínez-Zamora, Maria Angeles
Böke, Elvan
SpringerLink (Online service)

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Oocytes maintain ROS-free mitochondrial metabolism by suppressing complex I

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Other Objects (12)

Crosstalk between mitochondrial biogenesis and mitophagy to maintain mitochondrial homeostasis

Two mechanisms repress cyclin B1 translation to maintain prophase arrest in mouse oocytes

A pair of transporters controls mitochondrial Zn2+ levels to maintain mitochondrial homeostasis

Targeting macrophagic PIM-1 alleviates osteoarthritis by inhibiting NLRP3 inflammasome activation via suppressing mitochondrial ROS/Cl− efflux signaling pathway

Vimentin and Desmin Intermediate Filaments Maintain Mitochondrial Membrane Potential

Mitochondrial enzyme FAHD1 reduces ROS in osteosarcoma

Cryopreserved placental biopsies maintain mitochondrial activity for high-resolution respirometry

Basal Gp78-dependent mitophagy promotes mitochondrial health and limits mitochondrial ROS

CKB Promotes Mitochondrial ATP Production by Suppressing Permeability Transition Pore

PPTC7 maintains mitochondrial protein content by suppressing receptor-mediated mitophagy

Embryo development is impaired by sperm mitochondrial-derived ROS

Suppressing MTERF3 inhibits proliferation of human hepatocellular carcinoma via ROS-mediated p38 MAPK activation

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Two mechanisms repress cyclin B1 translation to maintain prophase arrest in mouse oocytes

A pair of transporters controls mitochondrial Zn2+ levels to maintain mitochondrial homeostasis

Targeting macrophagic PIM-1 alleviates osteoarthritis by inhibiting NLRP3 inflammasome activation via suppressing mitochondrial ROS/Cl− efflux signaling pathway

Vimentin and Desmin Intermediate Filaments Maintain Mitochondrial Membrane Potential

Mitochondrial enzyme FAHD1 reduces ROS in osteosarcoma

Cryopreserved placental biopsies maintain mitochondrial activity for high-resolution respirometry

Basal Gp78-dependent mitophagy promotes mitochondrial health and limits mitochondrial ROS

CKB Promotes Mitochondrial ATP Production by Suppressing Permeability Transition Pore

PPTC7 maintains mitochondrial protein content by suppressing receptor-mediated mitophagy

Embryo development is impaired by sperm mitochondrial-derived ROS

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Crosstalk between mitochondrial biogenesis and mitophagy to maintain mitochondrial homeostasis

Two mechanisms repress cyclin B1 translation to maintain prophase arrest in mouse oocytes

A pair of transporters controls mitochondrial Zn2+ levels to maintain mitochondrial homeostasis

Targeting macrophagic PIM-1 alleviates osteoarthritis by inhibiting NLRP3 inflammasome activation via suppressing mitochondrial ROS/Cl− efflux signaling pathway

Vimentin and Desmin Intermediate Filaments Maintain Mitochondrial Membrane Potential

Mitochondrial enzyme FAHD1 reduces ROS in osteosarcoma

Cryopreserved placental biopsies maintain mitochondrial activity for high-resolution respirometry

Basal Gp78-dependent mitophagy promotes mitochondrial health and limits mitochondrial ROS

CKB Promotes Mitochondrial ATP Production by Suppressing Permeability Transition Pore

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