Nrf2 activation drive macrophages polarization and cancer cell epithelial-mesenchymal transition during interaction
- Standort
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1478-811X
- Umfang
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Online-Ressource
- Sprache
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Englisch
- Anmerkungen
-
online resource.
- Erschienen in
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Nrf2 activation drive macrophages polarization and cancer cell epithelial-mesenchymal transition during interaction ; volume:16 ; number:1 ; day:4 ; month:9 ; year:2018 ; pages:1-12 ; date:12.2018
Cell communication and signaling ; 16, Heft 1 (4.9.2018), 1-12, 12.2018
- Urheber
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Feng, Rui
- Beteiligte Personen und Organisationen
-
Morine, Yuji
Ikemoto, Tetsuya
Imura, Satoru
Iwahashi, Shuichi
Saito, Yu
Shimada, Mitsuo
SpringerLink (Online service)
- DOI
-
10.1186/s12964-018-0262-x
- URN
-
urn:nbn:de:101:1-2018102222252353348977
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
15.08.2025, 07:31 MESZ
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Beteiligte
- Feng, Rui
- Morine, Yuji
- Ikemoto, Tetsuya
- Imura, Satoru
- Iwahashi, Shuichi
- Saito, Yu
- Shimada, Mitsuo
- SpringerLink (Online service)
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Nrf2 antioxidant pathway suppresses Numb-mediated epithelial–mesenchymal transition during pulmonary fibrosis
Intratumoral macrophages contribute to epithelial-mesenchymal transition in solid tumors
M2 macrophage polarization modulates epithelial-mesenchymal transition in cisplatin-induced tubulointerstitial fibrosis
5-Methoxyflavone alleviates LPS-mediated lung injury by promoting Nrf2-mediated the suppression of NOX4/TLR4 axis in bronchial epithelial cells and M1 polarization in macrophages
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Exosomes from high glucose-treated macrophages promote epithelial–mesenchymal transition of renal tubular epithelial cells via long non-coding RNAs
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Phloretin suppresses neuroinflammation by autophagy-mediated Nrf2 activation in macrophages
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