MiR-612 regulates invadopodia of hepatocellular carcinoma by HADHA-mediated lipid reprogramming
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1756-8722
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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MiR-612 regulates invadopodia of hepatocellular carcinoma by HADHA-mediated lipid reprogramming ; volume:13 ; number:1 ; day:7 ; month:2 ; year:2020 ; pages:1-19 ; date:12.2020
Journal of hematology & oncology ; 13, Heft 1 (7.2.2020), 1-19, 12.2020
- Creator
-
Liu, Yang
Lu, Li-Li
Wen, Duo
Liu, Dong-Li
Dong, Li-Li
Gao, Dong-Mei
Bian, Xin-Yu
Zhou, Jian
Fan, Jia
Wu, Wei-Zhong
- Contributor
-
SpringerLink (Online service)
- DOI
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10.1186/s13045-019-0841-3
- URN
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urn:nbn:de:101:1-2020071218011478277295
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:56 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Liu, Yang
- Lu, Li-Li
- Wen, Duo
- Liu, Dong-Li
- Dong, Li-Li
- Gao, Dong-Mei
- Bian, Xin-Yu
- Zhou, Jian
- Fan, Jia
- Wu, Wei-Zhong
- SpringerLink (Online service)
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Correction to: MiR-612 regulates invadopodia of hepatocellular carcinoma by HADHA-mediated lipid reprogramming
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Correction to: MiR-612 regulates invadopodia of hepatocellular carcinoma by HADHA-mediated lipid reprogramming
The evolution of the small x gluon TMD
Functional Silver‐Loaded Porous Composite Scaffold for Bone Tissue Bacterial Infection
DNMT1 mediates metabolic reprogramming induced by Epstein–Barr virus latent membrane protein 1 and reversed by grifolin in nasopharyngeal carcinoma
Determining the role of external beam radiotherapy in unresectable intrahepatic cholangiocarcinoma: a retrospective analysis of 84 patients
DCK is a promising prognostic biomarker and correlated with immune infiltrates in hepatocellular carcinoma
Microvascular invasion has limited clinical values in hepatocellular carcinoma patients at Barcelona Clinic Liver Cancer (BCLC) stages 0 or B
Association of preoperative EpCAM Circulating Tumor Cells and peripheral Treg cell levels with early recurrence of hepatocellular carcinoma following radical hepatic resection
Hypoxia-inducible factor-1 alpha, in association with inflammation, angiogenesis and MYC, is a critical prognostic factor in patients with HCC after surgery
Advances in clinical and basic research for hepatocellular carcinoma in China: a 2023 review
Risk factors affecting prognosis in metachronous liver metastases from WHO classification G1 and G2 gastroenteropancreatic neuroendocrine tumors after initial R0 surgical resection
Tumor-associated macrophages modulate resistance to oxaliplatin via inducing autophagy in hepatocellular carcinoma
Correction to: MiR-612 regulates invadopodia of hepatocellular carcinoma by HADHA-mediated lipid reprogramming
The evolution of the small x gluon TMD
Functional Silver‐Loaded Porous Composite Scaffold for Bone Tissue Bacterial Infection
DNMT1 mediates metabolic reprogramming induced by Epstein–Barr virus latent membrane protein 1 and reversed by grifolin in nasopharyngeal carcinoma
Determining the role of external beam radiotherapy in unresectable intrahepatic cholangiocarcinoma: a retrospective analysis of 84 patients
DCK is a promising prognostic biomarker and correlated with immune infiltrates in hepatocellular carcinoma
Microvascular invasion has limited clinical values in hepatocellular carcinoma patients at Barcelona Clinic Liver Cancer (BCLC) stages 0 or B
Association of preoperative EpCAM Circulating Tumor Cells and peripheral Treg cell levels with early recurrence of hepatocellular carcinoma following radical hepatic resection
Hypoxia-inducible factor-1 alpha, in association with inflammation, angiogenesis and MYC, is a critical prognostic factor in patients with HCC after surgery
Advances in clinical and basic research for hepatocellular carcinoma in China: a 2023 review
Risk factors affecting prognosis in metachronous liver metastases from WHO classification G1 and G2 gastroenteropancreatic neuroendocrine tumors after initial R0 surgical resection