Inhibition of basal-like breast cancer growth by FTY720 in combination with epidermal growth factor receptor kinase blockade
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1465-542X
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
Inhibition of basal-like breast cancer growth by FTY720 in combination with epidermal growth factor receptor kinase blockade ; volume:19 ; number:1 ; day:4 ; month:8 ; year:2017 ; pages:1-14 ; date:12.2017
Breast cancer research ; 19, Heft 1 (4.8.2017), 1-14, 12.2017
- Schlagwort
-
Krebs
Onkologie
- Urheber
-
Martin, Janet L.
- Beteiligte Personen und Organisationen
-
Julovi, Sohel M.
Lin, Mike Z.
de Silva, Hasanthi C.
Boyle, Frances M.
Baxter, Robert C.
SpringerLink (Online service)
- DOI
-
10.1186/s13058-017-0882-x
- URN
-
urn:nbn:de:1111-201710209915
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
- 15.08.2025, 07:27 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Martin, Janet L.
- Julovi, Sohel M.
- Lin, Mike Z.
- de Silva, Hasanthi C.
- Boyle, Frances M.
- Baxter, Robert C.
- SpringerLink (Online service)
Ähnliche Objekte (12)
Targeting the Transforming Growth Factor-β pathway inhibits human basal-like breast cancer metastasis
Feline mammary basal-like adenocarcinomas: a potential model for human triple-negative breast cancer (TNBC) with basal-like subtype
1,1-Bis(3'-indolyl)-1-(p-biphenyl)methane inhibits basal-like breast cancer growth in athymic nude mice
Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas
FAK activates AKT-mTOR signaling to promote the growth and progression of MMTV-Wnt1-driven basal-like mammary tumors
Weight loss reduces basal-like breast cancer through kinome reprogramming
Profiling and metaanalysis of epidermal keratinocytes responses to epidermal growth factor
Growth and biomechanics of plant epidermal cells
Targeting BCL-2–expressing basal-like breast cancer with BH3-mimetics
Basal-like breast cancer: molecular profiles, clinical features and survival outcomes
The CD44+/CD24-phenotype is enriched in basal-like breast tumors
Correction to: FAK activates AKT-mTOR signaling to promote the growth and progression of MMTV-Wnt1-driven basal-like mammary tumors
Targeting the Transforming Growth Factor-β pathway inhibits human basal-like breast cancer metastasis
Feline mammary basal-like adenocarcinomas: a potential model for human triple-negative breast cancer (TNBC) with basal-like subtype
1,1-Bis(3'-indolyl)-1-(p-biphenyl)methane inhibits basal-like breast cancer growth in athymic nude mice
Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas
FAK activates AKT-mTOR signaling to promote the growth and progression of MMTV-Wnt1-driven basal-like mammary tumors
Weight loss reduces basal-like breast cancer through kinome reprogramming
Profiling and metaanalysis of epidermal keratinocytes responses to epidermal growth factor
Growth and biomechanics of plant epidermal cells
Targeting BCL-2–expressing basal-like breast cancer with BH3-mimetics
Basal-like breast cancer: molecular profiles, clinical features and survival outcomes
The CD44+/CD24-phenotype is enriched in basal-like breast tumors
Correction to: FAK activates AKT-mTOR signaling to promote the growth and progression of MMTV-Wnt1-driven basal-like mammary tumors
Targeting the Transforming Growth Factor-β pathway inhibits human basal-like breast cancer metastasis
Feline mammary basal-like adenocarcinomas: a potential model for human triple-negative breast cancer (TNBC) with basal-like subtype
1,1-Bis(3'-indolyl)-1-(p-biphenyl)methane inhibits basal-like breast cancer growth in athymic nude mice
Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas
FAK activates AKT-mTOR signaling to promote the growth and progression of MMTV-Wnt1-driven basal-like mammary tumors
Weight loss reduces basal-like breast cancer through kinome reprogramming
Profiling and metaanalysis of epidermal keratinocytes responses to epidermal growth factor
Growth and biomechanics of plant epidermal cells
Targeting BCL-2–expressing basal-like breast cancer with BH3-mimetics
Basal-like breast cancer: molecular profiles, clinical features and survival outcomes
The CD44+/CD24-phenotype is enriched in basal-like breast tumors