TLR4 activation alters labile heme levels to regulate BACH1 and HO-1 expression in macrophages
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource
- Notes
-
Hannover, Medizinische Hochschule Hannover, Dissertation, 2019
- Keyword
-
Macrophages
Expression
Heme
- Event
-
Veröffentlichung
- (where)
-
Hannover
- (who)
-
Bibliothek der Medizinischen Hochschule Hannover
- (when)
-
2019
- Creator
- Contributor
- DOI
-
10.26068/mhhrpm/20191104-010
- URN
-
urn:nbn:de:101:1-2019121713252350143727
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 11:03 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Sudan, Kritika
- Immenschuh, Stephan
- Janciauskiene, Sabina
- Institut für Transfusionsmedizin
- Bibliothek der Medizinischen Hochschule Hannover
Time of origin
- 2019
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Labile Weltkonjunktur
Labile Weltwirtschaft
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Investigation of the procoagulant role of labile heme under hemolytic conditions
Labile heme aggravates renal inflammation and complement activation after ischemia reperfusion injury
Glyceraldehyde-3-phosphate dehydrogenase is a chaperone that allocates labile heme in cells
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Macrophages regulate healing-associated fibroblasts in diabetic wound
Sptlc2-derived sphinganine is indispensable for inflammatory TLR4 signaling in macrophages
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Heme activates TLR4-mediated inflammatory injury via MyD88/TRIF signaling pathway in intracerebral hemorrhage
Labile Weltkonjunktur
Labile Weltwirtschaft
Glutathione and the intracellular labile heme pool
Investigation of the procoagulant role of labile heme under hemolytic conditions
Labile heme aggravates renal inflammation and complement activation after ischemia reperfusion injury
Glyceraldehyde-3-phosphate dehydrogenase is a chaperone that allocates labile heme in cells
Labil
Macrophages regulate healing-associated fibroblasts in diabetic wound
Sptlc2-derived sphinganine is indispensable for inflammatory TLR4 signaling in macrophages
Bacterial vesicles block viral replication in macrophages via TLR4-TRIF-axis
WISP1 and TLR4 on Macrophages Contribute to Ventilator-Induced Lung Injury
Heme activates TLR4-mediated inflammatory injury via MyD88/TRIF signaling pathway in intracerebral hemorrhage
Labile Weltkonjunktur