Chronic Toxoplasma gondii infection enhances β-amyloid phagocytosis and clearance by recruited monocytes
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2051-5960
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Chronic Toxoplasma gondii infection enhances β-amyloid phagocytosis and clearance by recruited monocytes ; volume:4 ; number:1 ; day:16 ; month:3 ; year:2016 ; pages:1-19 ; date:12.2016
Acta Neuropathologica Communications ; 4, Heft 1 (16.3.2016), 1-19, 12.2016
- Creator
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Möhle, Luisa
- Contributor
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Israel, Nicole
Paarmann, Kristin
Krohn, Markus
Pietkiewicz, Sabine
Müller, Andreas Johann
Lavrik, Inna N.
Buguliskis, Jeffrey S.
Schott, Björn H.
Schlüter, Dirk
Gundelfinger, Eckart D.
Montag, Dirk
Seifert, Ulrike
Pahnke, Jens
Dunay, Ildikò Rita
SpringerLink (Online service)
- DOI
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10.1186/s40478-016-0293-8
- URN
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urn:nbn:de:1111-201603171931
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:49 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Möhle, Luisa
- Israel, Nicole
- Paarmann, Kristin
- Krohn, Markus
- Pietkiewicz, Sabine
- Müller, Andreas Johann
- Lavrik, Inna N.
- Buguliskis, Jeffrey S.
- Schott, Björn H.
- Schlüter, Dirk
- Gundelfinger, Eckart D.
- Montag, Dirk
- Seifert, Ulrike
- Pahnke, Jens
- Dunay, Ildikò Rita
- SpringerLink (Online service)
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Expression of area-specific M2-macrophage phenotype by recruited rat monocytes in duct-ligation pancreatitis
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Amphiregulin Regulates Phagocytosis-Induced Cell Death in Monocytes via EGFR and Matrix Metalloproteinases
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Amphiregulin Regulates Phagocytosis-Induced Cell Death in Monocytes via EGFR and the Bcl-2 Protein Family
Differential impact of zinc deficiency on phagocytosis, oxidative burst, and production of pro-inflammatory cytokines by human monocytes
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