In-depth analysis of alternative splicing landscape in multiple myeloma and potential role of dysregulated splicing factors
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2044-5385
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
-
In-depth analysis of alternative splicing landscape in multiple myeloma and potential role of dysregulated splicing factors ; volume:12 ; number:12 ; day:20 ; month:12 ; year:2022 ; pages:1-11 ; date:12.2022
Blood cancer journal ; 12, Heft 12 (20.12.2022), 1-11, 12.2022
- Creator
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Aktas Samur, Anil
Fulciniti, Mariateresa
Avet-Loiseau, Herve
Lopez, Michael A.
Derebail, Sanika
Corre, Jill
Minvielle, Stephane
Magrangeas, Florence
Moreau, Philippe
Anderson, Kenneth C.
Parmigiani, Giovanni
Samur, Mehmet K.
Munshi, Nikhil C.
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s41408-022-00759-6
- URN
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urn:nbn:de:101:1-2023030221024292812293
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:46 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Aktas Samur, Anil
- Fulciniti, Mariateresa
- Avet-Loiseau, Herve
- Lopez, Michael A.
- Derebail, Sanika
- Corre, Jill
- Minvielle, Stephane
- Magrangeas, Florence
- Moreau, Philippe
- Anderson, Kenneth C.
- Parmigiani, Giovanni
- Samur, Mehmet K.
- Munshi, Nikhil C.
- SpringerLink (Online service)
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The splicing machinery is dysregulated and represents a therapeutic vulnerability in breast cancer
Genome-wide identification and functional analysis of dysregulated alternative splicing profiles in sepsis
Transcriptome analysis of the parasite Encephalitozoon cuniculi: an in-depth examination of pre-mRNA splicing in a reduced eukaryote
Dysregulated splicing factor SF3B1 unveils a dual therapeutic vulnerability to target pancreatic cancer cells and cancer stem cells with an anti-splicing drug
RNA splicing and splicing regulator changes in prostate cancer pathology
Integrating many co-splicing networks to reconstruct splicing regulatory modules
Calculation of Splicing Potential from the Alternative Splicing Mutation Database
Molecular recognition of splicing factors involved in Fas alternative splicing
Mesenchymal stem cell contact promotes CCN1 splicing and transcription in myeloma cells
Mesenchymal stem cell contact promotes CCN1 splicing and transcription in myeloma cells
Dysregulated Rbfox2 produces aberrant splicing of CaV1.2 calcium channel in diabetes-induced cardiac hypertrophy
Function of splicing factor Tra2-beta1 in alternative splicing and translation
The splicing machinery is dysregulated and represents a therapeutic vulnerability in breast cancer
Genome-wide identification and functional analysis of dysregulated alternative splicing profiles in sepsis
Transcriptome analysis of the parasite Encephalitozoon cuniculi: an in-depth examination of pre-mRNA splicing in a reduced eukaryote
Dysregulated splicing factor SF3B1 unveils a dual therapeutic vulnerability to target pancreatic cancer cells and cancer stem cells with an anti-splicing drug
RNA splicing and splicing regulator changes in prostate cancer pathology
Integrating many co-splicing networks to reconstruct splicing regulatory modules
Calculation of Splicing Potential from the Alternative Splicing Mutation Database
Molecular recognition of splicing factors involved in Fas alternative splicing
Mesenchymal stem cell contact promotes CCN1 splicing and transcription in myeloma cells
Mesenchymal stem cell contact promotes CCN1 splicing and transcription in myeloma cells
Dysregulated Rbfox2 produces aberrant splicing of CaV1.2 calcium channel in diabetes-induced cardiac hypertrophy