Alternatively spliced ELAVL3 cryptic exon 4a causes ELAVL3 downregulation in ALS TDP-43 proteinopathy

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Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: 1 Online-Ressource.

Language: Englisch

Bibliographic citation: Alternatively spliced ELAVL3 cryptic exon 4a causes ELAVL3 downregulation in ALS TDP-43 proteinopathy ; volume:147 ; number:1 ; day:30 ; month:5 ; year:2024 ; pages:1-4 ; date:6.2024
Acta neuropathologica ; 147, Heft 1 (30.5.2024), 1-4, 6.2024

Creator: Costantino, Isabel
Meng, Alex
Ravits, John

Contributor: SpringerLink (Online service)

DOI: 10.1007/s00401-024-02732-y

URN: urn:nbn:de:101:1-2408090922214.801583317671

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:48 AM CEST

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Costantino, Isabel
Meng, Alex
Ravits, John
SpringerLink (Online service)

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TDP-43-regulated cryptic RNAs accumulate in Alzheimer’s disease brains

Tdp-43 cryptic exons are highly variable between cell types

Elevated nuclear TDP-43 induces constitutive exon skipping

Predicting mutually exclusive spliced exons based on exon length, splice site and reading frame conservation, and exon sequence homology

Tracking the evolution of alternatively spliced exons within the Dscam family

A phylogenetic generalized hidden Markov model for predicting alternatively spliced exons

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HDGFL2 cryptic proteins report presence of TDP-43 pathology in neurodegenerative diseases

TDP-43-regulated cryptic RNAs accumulate in Alzheimer’s disease brains

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Alternatively spliced ELAVL3 cryptic exon 4a causes ELAVL3 downregulation in ALS TDP-43 proteinopathy

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Other Objects (12)

Tdp-43 cryptic exons are highly variable between cell types

Elevated nuclear TDP-43 induces constitutive exon skipping

Predicting mutually exclusive spliced exons based on exon length, splice site and reading frame conservation, and exon sequence homology

Tracking the evolution of alternatively spliced exons within the Dscam family

A phylogenetic generalized hidden Markov model for predicting alternatively spliced exons

Quantitative analysis of cryptic splicing associated with TDP-43 depletion

Two alternatively-spliced human nebulin isoforms with either exon 143 or exon 144 and their developmental regulation

The era of cryptic exons: implications for ALS-FTD

Assessing the number of ancestral alternatively spliced exons in the human genome

Alternatively spliced exon regulates context-dependent MEF2D higher-order assembly during myogenesis

HDGFL2 cryptic proteins report presence of TDP-43 pathology in neurodegenerative diseases

TDP-43-regulated cryptic RNAs accumulate in Alzheimer’s disease brains

Tdp-43 cryptic exons are highly variable between cell types

Elevated nuclear TDP-43 induces constitutive exon skipping

Predicting mutually exclusive spliced exons based on exon length, splice site and reading frame conservation, and exon sequence homology

Tracking the evolution of alternatively spliced exons within the Dscam family

A phylogenetic generalized hidden Markov model for predicting alternatively spliced exons

Quantitative analysis of cryptic splicing associated with TDP-43 depletion

Two alternatively-spliced human nebulin isoforms with either exon 143 or exon 144 and their developmental regulation

The era of cryptic exons: implications for ALS-FTD

Assessing the number of ancestral alternatively spliced exons in the human genome

Alternatively spliced exon regulates context-dependent MEF2D higher-order assembly during myogenesis

HDGFL2 cryptic proteins report presence of TDP-43 pathology in neurodegenerative diseases

TDP-43-regulated cryptic RNAs accumulate in Alzheimer’s disease brains

Tdp-43 cryptic exons are highly variable between cell types

Elevated nuclear TDP-43 induces constitutive exon skipping

Predicting mutually exclusive spliced exons based on exon length, splice site and reading frame conservation, and exon sequence homology

Tracking the evolution of alternatively spliced exons within the Dscam family

A phylogenetic generalized hidden Markov model for predicting alternatively spliced exons

Quantitative analysis of cryptic splicing associated with TDP-43 depletion

Two alternatively-spliced human nebulin isoforms with either exon 143 or exon 144 and their developmental regulation

The era of cryptic exons: implications for ALS-FTD

Assessing the number of ancestral alternatively spliced exons in the human genome

Alternatively spliced exon regulates context-dependent MEF2D higher-order assembly during myogenesis

HDGFL2 cryptic proteins report presence of TDP-43 pathology in neurodegenerative diseases

TDP-43-regulated cryptic RNAs accumulate in Alzheimer’s disease brains

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