The downregulation of Mcl-1 via USP9X inhibition sensitizes solid tumors to Bcl-xl inhibition

to related objects

Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1471-2407

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: The downregulation of Mcl-1 via USP9X inhibition sensitizes solid tumors to Bcl-xl inhibition ; volume:12 ; number:1 ; day:21 ; month:11 ; year:2012 ; pages:1-12 ; date:12.2012
BMC cancer ; 12, Heft 1 (21.11.2012), 1-12, 12.2012

Creator: Peddaboina, Chander

Contributor: Jupiter, Daniel
Fletcher, Steven
Yap, Jeremy L.
Rai, Arun
Tobin, Richard P.
Jiang, Weihua
Rascoe, Philip
Rogers, M Karen Newell
Smythe, W Roy
Cao, Xiaobo
SpringerLink (Online service)

DOI: 10.1186/1471-2407-12-541

URN: urn:nbn:de:1111-20160304527

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:37 AM CEST

Data provider

This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.

Show original at data provider

Associated

Peddaboina, Chander
Jupiter, Daniel
Fletcher, Steven
Yap, Jeremy L.
Rai, Arun
Tobin, Richard P.
Jiang, Weihua
Rascoe, Philip
Rogers, M Karen Newell
Smythe, W Roy
Cao, Xiaobo
SpringerLink (Online service)

Other Objects (12)

Dual inhibition of anti-apoptotic proteins BCL-XL and MCL-1 enhances cytotoxicity of Nasopharyngeal carcinoma cells

BH3-only proteins are dispensable for apoptosis induced by pharmacological inhibition of both MCL-1 and BCL-XL

Hochschulschrift

Targeting antiapoptotic proteins Bcl-xL and Mcl-1 in cholangiocarcinoma

Calcium signals inhibition sensitizes ovarian carcinoma cells to anti-Bcl-xL strategies through Mcl-1 down-regulation

Photodynamic therapy combined with Bcl-2/Bcl-xL inhibition increases the Noxa/Mcl-1 ratio independent of usp9X and synergistically enhances apoptosis in glioblastoma

Rolle von Bcl-2, Bcl-xl und Mcl-1 für die Apoptoseresistenz und Chemoresistenz des Pankreaskarzinoms

BCL-XL and MCL-1 are the key BCL-2 family proteins in melanoma cell survival

Bcl-xL inhibition enhances Dinaciclib-induced cell death in soft-tissue sarcomas

Hochschulschrift

Transient apoptosis inhibition by Bcl-xL overexpression during hematopoietic stem cell transplantation

Dual Inhibition of Bcl-2/Bcl-xL and XPO1 is synthetically lethal in glioblastoma model systems

Inhibition of Bcl-2/Bcl-xL and c-MET causes synthetic lethality in model systems of glioblastoma

Inhibition of cyclin E1 sensitizes hepatocellular carcinoma cells to regorafenib by mcl-1 suppression

Dual inhibition of anti-apoptotic proteins BCL-XL and MCL-1 enhances cytotoxicity of Nasopharyngeal carcinoma cells

BH3-only proteins are dispensable for apoptosis induced by pharmacological inhibition of both MCL-1 and BCL-XL

Hochschulschrift

Targeting antiapoptotic proteins Bcl-xL and Mcl-1 in cholangiocarcinoma

Calcium signals inhibition sensitizes ovarian carcinoma cells to anti-Bcl-xL strategies through Mcl-1 down-regulation

Photodynamic therapy combined with Bcl-2/Bcl-xL inhibition increases the Noxa/Mcl-1 ratio independent of usp9X and synergistically enhances apoptosis in glioblastoma

Rolle von Bcl-2, Bcl-xl und Mcl-1 für die Apoptoseresistenz und Chemoresistenz des Pankreaskarzinoms

BCL-XL and MCL-1 are the key BCL-2 family proteins in melanoma cell survival

Bcl-xL inhibition enhances Dinaciclib-induced cell death in soft-tissue sarcomas

Hochschulschrift

Transient apoptosis inhibition by Bcl-xL overexpression during hematopoietic stem cell transplantation

Dual Inhibition of Bcl-2/Bcl-xL and XPO1 is synthetically lethal in glioblastoma model systems

Inhibition of Bcl-2/Bcl-xL and c-MET causes synthetic lethality in model systems of glioblastoma

Inhibition of cyclin E1 sensitizes hepatocellular carcinoma cells to regorafenib by mcl-1 suppression

Dual inhibition of anti-apoptotic proteins BCL-XL and MCL-1 enhances cytotoxicity of Nasopharyngeal carcinoma cells

BH3-only proteins are dispensable for apoptosis induced by pharmacological inhibition of both MCL-1 and BCL-XL

Hochschulschrift

Targeting antiapoptotic proteins Bcl-xL and Mcl-1 in cholangiocarcinoma

Calcium signals inhibition sensitizes ovarian carcinoma cells to anti-Bcl-xL strategies through Mcl-1 down-regulation

Photodynamic therapy combined with Bcl-2/Bcl-xL inhibition increases the Noxa/Mcl-1 ratio independent of usp9X and synergistically enhances apoptosis in glioblastoma

Rolle von Bcl-2, Bcl-xl und Mcl-1 für die Apoptoseresistenz und Chemoresistenz des Pankreaskarzinoms

BCL-XL and MCL-1 are the key BCL-2 family proteins in melanoma cell survival

Bcl-xL inhibition enhances Dinaciclib-induced cell death in soft-tissue sarcomas

Hochschulschrift

Transient apoptosis inhibition by Bcl-xL overexpression during hematopoietic stem cell transplantation

Dual Inhibition of Bcl-2/Bcl-xL and XPO1 is synthetically lethal in glioblastoma model systems

Inhibition of Bcl-2/Bcl-xL and c-MET causes synthetic lethality in model systems of glioblastoma

Inhibition of cyclin E1 sensitizes hepatocellular carcinoma cells to regorafenib by mcl-1 suppression

Cultural heritage institutions wishing to register will find more information here.

Fields marked * need to be filled in.

Username*

Please enter your username

Email*

Please enter your email address

Please do not fill this field

First name

Last name

Password*

Please enter your password

Confirm password*

Please enter the same password

I have read the terms of use and the privacy policy for the collection of personal data and accept them. *

This field is required.

I would like to subscribe to the newsletter of the Deutsche Digitale Bibliothek. See newsletter subscription info.

Account created

Your "My DDB" account has been successfully created. Before you can log in to your account, you must click the confirmation link in the message we just sent to the email address you provided.

The downregulation of Mcl-1 via USP9X inhibition sensitizes solid tumors to Bcl-xl inhibition

Object Details

References and Relationships

Contributors, Places and Time

Further information

Data provider

Associated

Other Objects (12)

Dual inhibition of anti-apoptotic proteins BCL-XL and MCL-1 enhances cytotoxicity of Nasopharyngeal carcinoma cells

BH3-only proteins are dispensable for apoptosis induced by pharmacological inhibition of both MCL-1 and BCL-XL

Targeting antiapoptotic proteins Bcl-xL and Mcl-1 in cholangiocarcinoma

Calcium signals inhibition sensitizes ovarian carcinoma cells to anti-Bcl-xL strategies through Mcl-1 down-regulation

Photodynamic therapy combined with Bcl-2/Bcl-xL inhibition increases the Noxa/Mcl-1 ratio independent of usp9X and synergistically enhances apoptosis in glioblastoma

Rolle von Bcl-2, Bcl-xl und Mcl-1 für die Apoptoseresistenz und Chemoresistenz des Pankreaskarzinoms

BCL-XL and MCL-1 are the key BCL-2 family proteins in melanoma cell survival

Bcl-xL inhibition enhances Dinaciclib-induced cell death in soft-tissue sarcomas

Transient apoptosis inhibition by Bcl-xL overexpression during hematopoietic stem cell transplantation

Dual Inhibition of Bcl-2/Bcl-xL and XPO1 is synthetically lethal in glioblastoma model systems

Inhibition of Bcl-2/Bcl-xL and c-MET causes synthetic lethality in model systems of glioblastoma

Inhibition of cyclin E1 sensitizes hepatocellular carcinoma cells to regorafenib by mcl-1 suppression

Dual inhibition of anti-apoptotic proteins BCL-XL and MCL-1 enhances cytotoxicity of Nasopharyngeal carcinoma cells

BH3-only proteins are dispensable for apoptosis induced by pharmacological inhibition of both MCL-1 and BCL-XL

Targeting antiapoptotic proteins Bcl-xL and Mcl-1 in cholangiocarcinoma

Calcium signals inhibition sensitizes ovarian carcinoma cells to anti-Bcl-xL strategies through Mcl-1 down-regulation

Photodynamic therapy combined with Bcl-2/Bcl-xL inhibition increases the Noxa/Mcl-1 ratio independent of usp9X and synergistically enhances apoptosis in glioblastoma

Rolle von Bcl-2, Bcl-xl und Mcl-1 für die Apoptoseresistenz und Chemoresistenz des Pankreaskarzinoms

BCL-XL and MCL-1 are the key BCL-2 family proteins in melanoma cell survival

Bcl-xL inhibition enhances Dinaciclib-induced cell death in soft-tissue sarcomas

Transient apoptosis inhibition by Bcl-xL overexpression during hematopoietic stem cell transplantation

Dual Inhibition of Bcl-2/Bcl-xL and XPO1 is synthetically lethal in glioblastoma model systems

Inhibition of Bcl-2/Bcl-xL and c-MET causes synthetic lethality in model systems of glioblastoma

Inhibition of cyclin E1 sensitizes hepatocellular carcinoma cells to regorafenib by mcl-1 suppression

Dual inhibition of anti-apoptotic proteins BCL-XL and MCL-1 enhances cytotoxicity of Nasopharyngeal carcinoma cells

BH3-only proteins are dispensable for apoptosis induced by pharmacological inhibition of both MCL-1 and BCL-XL

Targeting antiapoptotic proteins Bcl-xL and Mcl-1 in cholangiocarcinoma

Calcium signals inhibition sensitizes ovarian carcinoma cells to anti-Bcl-xL strategies through Mcl-1 down-regulation

Photodynamic therapy combined with Bcl-2/Bcl-xL inhibition increases the Noxa/Mcl-1 ratio independent of usp9X and synergistically enhances apoptosis in glioblastoma

Rolle von Bcl-2, Bcl-xl und Mcl-1 für die Apoptoseresistenz und Chemoresistenz des Pankreaskarzinoms

BCL-XL and MCL-1 are the key BCL-2 family proteins in melanoma cell survival

Bcl-xL inhibition enhances Dinaciclib-induced cell death in soft-tissue sarcomas

Transient apoptosis inhibition by Bcl-xL overexpression during hematopoietic stem cell transplantation

Dual Inhibition of Bcl-2/Bcl-xL and XPO1 is synthetically lethal in glioblastoma model systems

Inhibition of Bcl-2/Bcl-xL and c-MET causes synthetic lethality in model systems of glioblastoma

Inhibition of cyclin E1 sensitizes hepatocellular carcinoma cells to regorafenib by mcl-1 suppression

Related objects

Reset password