Hypoxia Suppresses TGF-B1-Induced Cardiac Myocyte Myofibroblast Transformation by Inhibiting Smad2/3 and Rhoa Signaling Pathways
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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Online-Ressource
- Language
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Englisch
- Bibliographic citation
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Hypoxia Suppresses TGF-B1-Induced Cardiac Myocyte Myofibroblast Transformation by Inhibiting Smad2/3 and Rhoa Signaling Pathways ; volume:45 ; number:1 ; year:2018 ; pages:250-257
Cellular physiology and biochemistry ; 45, Heft 1 (2018), 250-257
- Creator
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Yan, Zhankui
Shen, Daifei
Liao, Jilin
Zhang, Yanmei
Chen, Yicun
Shi, Ganggang
Gao, Fenfei
- DOI
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10.1159/000486771
- URN
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urn:nbn:de:101:1-2018021418166
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:45 AM CEST
Data provider
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Yan, Zhankui
- Shen, Daifei
- Liao, Jilin
- Zhang, Yanmei
- Chen, Yicun
- Shi, Ganggang
- Gao, Fenfei
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SMAD2/3 mediate oncogenic effects of TGF-β in the absence of SMAD4
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TGF-β promotes pericyte-myofibroblast transition in subretinal fibrosis through the Smad2/3 and Akt/mTOR pathways
Adipose-derived stem cell exosomes attenuates myofibroblast transformation via inhibiting autophagy through TGF-β/Smad2 axis in oral submucosal fibrosis
TSC1 is a component of the TGF-β-Smad2-3 [TGF-beta-Smad2-3] pathway and mediates insulin-Akt-TGF-β [insulin-Akt-TGF-beta] crosstalk
RhoA suppresses pseudorabies virus replication in vitro
ACSL4 accelerates osteosarcoma progression via modulating TGF-β/Smad2 signaling pathway
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Smurf2 suppresses the metastasis of hepatocellular carcinoma via ubiquitin degradation of Smad2
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Unterschiede in der TGF-β-vermittelten [TGF-beta-vermittelten] Signaltransduktion in aktivierten hepatischen Sternzellen unterhalb der Smad2-Phosphorylierung
Unterschiede in der TGF-β-vermittelten [TGF-beta-vermittelten] Signaltransduktion in aktivierten hepatischen Sternzellen unterhalb der Smad2-Phosphorylierung
SMAD2/3 mediate oncogenic effects of TGF-β in the absence of SMAD4
BMP3 suppresses colon tumorigenesis via ActRIIB/SMAD2-dependent and TAK1/JNK signaling pathways
TGF-β promotes pericyte-myofibroblast transition in subretinal fibrosis through the Smad2/3 and Akt/mTOR pathways
Adipose-derived stem cell exosomes attenuates myofibroblast transformation via inhibiting autophagy through TGF-β/Smad2 axis in oral submucosal fibrosis
TSC1 is a component of the TGF-β-Smad2-3 [TGF-beta-Smad2-3] pathway and mediates insulin-Akt-TGF-β [insulin-Akt-TGF-beta] crosstalk
RhoA suppresses pseudorabies virus replication in vitro
ACSL4 accelerates osteosarcoma progression via modulating TGF-β/Smad2 signaling pathway
Creg1 Regulates Erythroid Development via TGF‐β/Smad2‐Klf1 Axis in Zebrafish
Smurf2 suppresses the metastasis of hepatocellular carcinoma via ubiquitin degradation of Smad2
Smurf2 suppresses the metastasis of hepatocellular carcinoma via ubiquitin degradation of Smad2
Unterschiede in der TGF-β-vermittelten [TGF-beta-vermittelten] Signaltransduktion in aktivierten hepatischen Sternzellen unterhalb der Smad2-Phosphorylierung
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