G9a/DNMT1 co-targeting inhibits non-small cell lung cancer growth and reprograms tumor cells to respond to cancer-drugs through SCARA5 and AOX1

to related objects

Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: 1 Online-Ressource.

Language: Englisch

Bibliographic citation: G9a/DNMT1 co-targeting inhibits non-small cell lung cancer growth and reprograms tumor cells to respond to cancer-drugs through SCARA5 and AOX1 ; volume:15 ; number:11 ; day:2 ; month:11 ; year:2024 ; pages:1-16 ; date:11.2024
Cell death & disease ; 15, Heft 11 (2.11.2024), 1-16, 11.2024

Creator: Exposito, Francisco
Redrado, Miriam
Serrano, Diego
Calabuig-Fariñas, Silvia
Bao-Caamano, Aida
Gallach, Sandra
Jantus-Lewintre, Eloisa
Diaz-Lagares, Angel
Rodriguez-Casanova, Aitor
Sandoval, Juan
San Jose-Eneriz, Edurne
Garcia, Javier
Redin, Esther
Senent, Yaiza
Leon, Sergio
Pio, Ruben
Lopez, Rafael
Oyarzabal, Julen
Pineda-Lucena, Antonio
Agirre, Xabier
Montuenga, Luis M.
Prosper, Felipe
Calvo, Alfonso

Contributor: SpringerLink (Online service)

DOI: 10.1038/s41419-024-07156-w

URN: urn:nbn:de:101:1-2501212124246.040862411207

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:32 AM CEST

Data provider

This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.

Show original at data provider

Associated

Exposito, Francisco
Redrado, Miriam
Serrano, Diego
Calabuig-Fariñas, Silvia
Bao-Caamano, Aida
Gallach, Sandra
Jantus-Lewintre, Eloisa
Diaz-Lagares, Angel
Rodriguez-Casanova, Aitor
Sandoval, Juan
San Jose-Eneriz, Edurne
Garcia, Javier
Redin, Esther
Senent, Yaiza
Leon, Sergio
Pio, Ruben
Lopez, Rafael
Oyarzabal, Julen
Pineda-Lucena, Antonio
Agirre, Xabier
Montuenga, Luis M.
Prosper, Felipe
Calvo, Alfonso
SpringerLink (Online service)

Other Objects (12)

The DNMT1-associated lincRNA DACOR1 reprograms genome-wide DNA methylation in colon cancer

Crosslinking of Ly6a metabolically reprograms CD8 T cells for cancer immunotherapy

Cervical cancer-produced neuromedin-B reprograms Schwann cells to initiate perineural invasion

S-allylcysteine suppresses ovarian cancer cell proliferation by DNA methylation through DNMT1

Cholesterol reprograms glucose and lipid metabolism to promote proliferation in colon cancer cells

Hinokitiol induces DNA demethylation via DNMT1 and UHRF1 inhibition in colon cancer cells

Correction: Cholesterol reprograms glucose and lipid metabolism to promote proliferation in colon cancer cells

Tamoxifen mechanically reprograms the tumor microenvironment via HIF ‐1A and reduces cancer cell survival

STON2 negatively modulates stem-like properties in ovarian cancer cells via DNMT1/MUC1 pathway

Mahanine restores RASSF1A expression by down-regulating DNMT1 and DNMT3B in prostate cancer cells

NARFL deficiency caused mitochondrial dysfunction in lung cancer cells by HIF-1α–DNMT1 axis

NNMT‐DNMT1 Axis is Essential for Maintaining Cancer Cell Sensitivity to Oxidative Phosphorylation Inhibition

The DNMT1-associated lincRNA DACOR1 reprograms genome-wide DNA methylation in colon cancer

Crosslinking of Ly6a metabolically reprograms CD8 T cells for cancer immunotherapy

Cervical cancer-produced neuromedin-B reprograms Schwann cells to initiate perineural invasion

S-allylcysteine suppresses ovarian cancer cell proliferation by DNA methylation through DNMT1

Cholesterol reprograms glucose and lipid metabolism to promote proliferation in colon cancer cells

Hinokitiol induces DNA demethylation via DNMT1 and UHRF1 inhibition in colon cancer cells

Correction: Cholesterol reprograms glucose and lipid metabolism to promote proliferation in colon cancer cells

Tamoxifen mechanically reprograms the tumor microenvironment via HIF ‐1A and reduces cancer cell survival

STON2 negatively modulates stem-like properties in ovarian cancer cells via DNMT1/MUC1 pathway

Mahanine restores RASSF1A expression by down-regulating DNMT1 and DNMT3B in prostate cancer cells

NARFL deficiency caused mitochondrial dysfunction in lung cancer cells by HIF-1α–DNMT1 axis

NNMT‐DNMT1 Axis is Essential for Maintaining Cancer Cell Sensitivity to Oxidative Phosphorylation Inhibition

The DNMT1-associated lincRNA DACOR1 reprograms genome-wide DNA methylation in colon cancer

Crosslinking of Ly6a metabolically reprograms CD8 T cells for cancer immunotherapy

Cervical cancer-produced neuromedin-B reprograms Schwann cells to initiate perineural invasion

S-allylcysteine suppresses ovarian cancer cell proliferation by DNA methylation through DNMT1

Cholesterol reprograms glucose and lipid metabolism to promote proliferation in colon cancer cells

Hinokitiol induces DNA demethylation via DNMT1 and UHRF1 inhibition in colon cancer cells

Correction: Cholesterol reprograms glucose and lipid metabolism to promote proliferation in colon cancer cells

Tamoxifen mechanically reprograms the tumor microenvironment via HIF ‐1A and reduces cancer cell survival

STON2 negatively modulates stem-like properties in ovarian cancer cells via DNMT1/MUC1 pathway

Mahanine restores RASSF1A expression by down-regulating DNMT1 and DNMT3B in prostate cancer cells

NARFL deficiency caused mitochondrial dysfunction in lung cancer cells by HIF-1α–DNMT1 axis

NNMT‐DNMT1 Axis is Essential for Maintaining Cancer Cell Sensitivity to Oxidative Phosphorylation Inhibition

Cultural heritage institutions wishing to register will find more information here.

Fields marked * need to be filled in.

Username*

Please enter your username

Email*

Please enter your email address

Please do not fill this field

First name

Last name

Password*

Please enter your password

Confirm password*

Please enter the same password

I have read the terms of use and the privacy policy for the collection of personal data and accept them. *

This field is required.

I would like to subscribe to the newsletter of the Deutsche Digitale Bibliothek. See newsletter subscription info.

Account created

Your "My DDB" account has been successfully created. Before you can log in to your account, you must click the confirmation link in the message we just sent to the email address you provided.

G9a/DNMT1 co-targeting inhibits non-small cell lung cancer growth and reprograms tumor cells to respond to cancer-drugs through SCARA5 and AOX1

Object Details

References and Relationships

Contributors, Places and Time

Further information

Data provider

Associated

Other Objects (12)

The DNMT1-associated lincRNA DACOR1 reprograms genome-wide DNA methylation in colon cancer

Crosslinking of Ly6a metabolically reprograms CD8 T cells for cancer immunotherapy

Cervical cancer-produced neuromedin-B reprograms Schwann cells to initiate perineural invasion

S-allylcysteine suppresses ovarian cancer cell proliferation by DNA methylation through DNMT1

Cholesterol reprograms glucose and lipid metabolism to promote proliferation in colon cancer cells

Hinokitiol induces DNA demethylation via DNMT1 and UHRF1 inhibition in colon cancer cells

Correction: Cholesterol reprograms glucose and lipid metabolism to promote proliferation in colon cancer cells

Tamoxifen mechanically reprograms the tumor microenvironment via HIF ‐1A and reduces cancer cell survival

STON2 negatively modulates stem-like properties in ovarian cancer cells via DNMT1/MUC1 pathway

Mahanine restores RASSF1A expression by down-regulating DNMT1 and DNMT3B in prostate cancer cells

NARFL deficiency caused mitochondrial dysfunction in lung cancer cells by HIF-1α–DNMT1 axis

NNMT‐DNMT1 Axis is Essential for Maintaining Cancer Cell Sensitivity to Oxidative Phosphorylation Inhibition

The DNMT1-associated lincRNA DACOR1 reprograms genome-wide DNA methylation in colon cancer

Crosslinking of Ly6a metabolically reprograms CD8 T cells for cancer immunotherapy

Cervical cancer-produced neuromedin-B reprograms Schwann cells to initiate perineural invasion

S-allylcysteine suppresses ovarian cancer cell proliferation by DNA methylation through DNMT1

Cholesterol reprograms glucose and lipid metabolism to promote proliferation in colon cancer cells

Hinokitiol induces DNA demethylation via DNMT1 and UHRF1 inhibition in colon cancer cells

Correction: Cholesterol reprograms glucose and lipid metabolism to promote proliferation in colon cancer cells

Tamoxifen mechanically reprograms the tumor microenvironment via HIF ‐1A and reduces cancer cell survival

STON2 negatively modulates stem-like properties in ovarian cancer cells via DNMT1/MUC1 pathway

Mahanine restores RASSF1A expression by down-regulating DNMT1 and DNMT3B in prostate cancer cells

NARFL deficiency caused mitochondrial dysfunction in lung cancer cells by HIF-1α–DNMT1 axis

NNMT‐DNMT1 Axis is Essential for Maintaining Cancer Cell Sensitivity to Oxidative Phosphorylation Inhibition

The DNMT1-associated lincRNA DACOR1 reprograms genome-wide DNA methylation in colon cancer

Crosslinking of Ly6a metabolically reprograms CD8 T cells for cancer immunotherapy

Cervical cancer-produced neuromedin-B reprograms Schwann cells to initiate perineural invasion

S-allylcysteine suppresses ovarian cancer cell proliferation by DNA methylation through DNMT1

Cholesterol reprograms glucose and lipid metabolism to promote proliferation in colon cancer cells

Hinokitiol induces DNA demethylation via DNMT1 and UHRF1 inhibition in colon cancer cells

Correction: Cholesterol reprograms glucose and lipid metabolism to promote proliferation in colon cancer cells

Tamoxifen mechanically reprograms the tumor microenvironment via HIF ‐1A and reduces cancer cell survival

STON2 negatively modulates stem-like properties in ovarian cancer cells via DNMT1/MUC1 pathway

Mahanine restores RASSF1A expression by down-regulating DNMT1 and DNMT3B in prostate cancer cells

NARFL deficiency caused mitochondrial dysfunction in lung cancer cells by HIF-1α–DNMT1 axis

NNMT‐DNMT1 Axis is Essential for Maintaining Cancer Cell Sensitivity to Oxidative Phosphorylation Inhibition

Related objects

Reset password