High Glucose-Induced Podocyte Injury Involves Activation of Mammalian Target of Rapamycin (mTOR)-Induced Endoplasmic Reticulum (ER) Stress
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Erschienen in
-
High Glucose-Induced Podocyte Injury Involves Activation of Mammalian Target of Rapamycin (mTOR)-Induced Endoplasmic Reticulum (ER) Stress ; volume:45 ; number:6 ; year:2018 ; pages:2431-2443
Cellular physiology and biochemistry ; 45, Heft 6 (2018), 2431-2443
- Urheber
-
Lei, Jie
Zhao, Lei
Zhang, Yujing
Wu, Yanfeng
Liu, Yanbo
- DOI
-
10.1159/000488231
- URN
-
urn:nbn:de:101:1-201805032084
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
14.08.2025, 10:51 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Lei, Jie
- Zhao, Lei
- Zhang, Yujing
- Wu, Yanfeng
- Liu, Yanbo
Ähnliche Objekte (12)
Rapamycin attenuated podocyte apoptosis via upregulation of nestin in Ang II-induced podocyte injury
The protective effect and mechanism of catalpol on high glucose-induced podocyte injury
Oryeongsan suppressed high glucose-induced mesangial fibrosis
Fructose impairs glucose-induced hepatic triglyceride synthesis
Fenofibrate reduces glucose-induced barrier dysfunction in feline enteroids
Podocyte specific deletion of PKM2 ameliorates LPS-induced podocyte injury through beta-catenin
[Gly14]-Humanin ameliorates high glucose-induced endothelial senescence via SIRT6
Podocyte-specific Rac1 deficiency ameliorates podocyte damage and proteinuria in STZ-induced diabetic nephropathy in mice
Fructose-induced stress signaling in the liver involves methylglyoxal
Rapamycin sensitizes T-ALL cells to dexamethasone-induced apoptosis
Estradiol Prevents High Glucose-Induced β-cell Apoptosis by Decreased BTG2 Expression
Rapamycin Treatment Reduces Acute Myocarditis Induced by Trypanosoma cruzi Infection
Rapamycin attenuated podocyte apoptosis via upregulation of nestin in Ang II-induced podocyte injury
The protective effect and mechanism of catalpol on high glucose-induced podocyte injury
Oryeongsan suppressed high glucose-induced mesangial fibrosis
Fructose impairs glucose-induced hepatic triglyceride synthesis
Fenofibrate reduces glucose-induced barrier dysfunction in feline enteroids
Podocyte specific deletion of PKM2 ameliorates LPS-induced podocyte injury through beta-catenin
[Gly14]-Humanin ameliorates high glucose-induced endothelial senescence via SIRT6
Podocyte-specific Rac1 deficiency ameliorates podocyte damage and proteinuria in STZ-induced diabetic nephropathy in mice
Fructose-induced stress signaling in the liver involves methylglyoxal
Rapamycin sensitizes T-ALL cells to dexamethasone-induced apoptosis
Estradiol Prevents High Glucose-Induced β-cell Apoptosis by Decreased BTG2 Expression
Rapamycin Treatment Reduces Acute Myocarditis Induced by Trypanosoma cruzi Infection
Rapamycin attenuated podocyte apoptosis via upregulation of nestin in Ang II-induced podocyte injury
The protective effect and mechanism of catalpol on high glucose-induced podocyte injury
Oryeongsan suppressed high glucose-induced mesangial fibrosis
Fructose impairs glucose-induced hepatic triglyceride synthesis
Fenofibrate reduces glucose-induced barrier dysfunction in feline enteroids
Podocyte specific deletion of PKM2 ameliorates LPS-induced podocyte injury through beta-catenin
[Gly14]-Humanin ameliorates high glucose-induced endothelial senescence via SIRT6
Podocyte-specific Rac1 deficiency ameliorates podocyte damage and proteinuria in STZ-induced diabetic nephropathy in mice
Fructose-induced stress signaling in the liver involves methylglyoxal
Rapamycin sensitizes T-ALL cells to dexamethasone-induced apoptosis
Estradiol Prevents High Glucose-Induced β-cell Apoptosis by Decreased BTG2 Expression