Gingerol suppresses sepsis-induced acute kidney injury by modulating methylsulfonylmethane and dimethylamine production

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 2045-2322

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Gingerol suppresses sepsis-induced acute kidney injury by modulating methylsulfonylmethane and dimethylamine production ; volume:8 ; number:1 ; day:14 ; month:8 ; year:2018 ; pages:1-10 ; date:12.2018
Scientific reports ; 8, Heft 1 (14.8.2018), 1-10, 12.2018

Creator: Rodrigues, Francisco Adelvane de Paulo

Contributor: Santos, Alan Diego da Conceição
Medeiros, Pedro Henrique Quintela Soares de
Prata, Mara de Moura Gondim
Santos, Tailane Caína de Souza
Silva, James Almada da
Brito, Gerly Anne de Castro
Santos, Armênio Aguiar dos
Silveira, Edilberto Rocha
Lima, Aldo Ângelo Moreira
Havt, Alexandre
SpringerLink (Online service)

DOI: 10.1038/s41598-018-30522-6

URN: urn:nbn:de:101:1-2018091411380692484815

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:24 AM CEST

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Associated

Rodrigues, Francisco Adelvane de Paulo
Santos, Alan Diego da Conceição
Medeiros, Pedro Henrique Quintela Soares de
Prata, Mara de Moura Gondim
Santos, Tailane Caína de Souza
Silva, James Almada da
Brito, Gerly Anne de Castro
Santos, Armênio Aguiar dos
Silveira, Edilberto Rocha
Lima, Aldo Ângelo Moreira
Havt, Alexandre
SpringerLink (Online service)

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Pathophysiology of sepsis-induced myocardial dysfunction

Sepsis-induced coagulopathy (SIC) in the management of sepsis

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Gingerol suppresses sepsis-induced acute kidney injury by modulating methylsulfonylmethane and dimethylamine production

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Other Objects (12)

Molecular differences in susceptibility of the kidney to sepsis-induced kidney injury

Sepsis-induced erythrocytes apoptosis

Modulating Nrf-2/HO-1, apoptosis and oxidative stress signaling pathways by gabapentin ameliorates sepsis-induced acute kidney injury

Pathophysiology of sepsis-induced myocardial dysfunction

Sepsis-induced coagulopathy (SIC) in the management of sepsis

The role of neutrophils in ischemia/reperfusion- and sepsis-induced acute kidney injury

Allicin ameliorates sepsis-induced acute kidney injury through Nrf2/HO-1 signaling pathway

Sepsis-induced myocardial dysfunction: pathophysiology and management

Gene signature for the prediction of the trajectories of sepsis-induced acute kidney injury

Sepsis-induced cardiac dysfunction and β-adrenergic blockade therapy for sepsis

PLK1 protects against sepsis-induced intestinal barrier dysfunction

Doxycycline protects against sepsis-induced endothelial glycocalyx shedding

Molecular differences in susceptibility of the kidney to sepsis-induced kidney injury

Sepsis-induced erythrocytes apoptosis

Modulating Nrf-2/HO-1, apoptosis and oxidative stress signaling pathways by gabapentin ameliorates sepsis-induced acute kidney injury

Pathophysiology of sepsis-induced myocardial dysfunction

Sepsis-induced coagulopathy (SIC) in the management of sepsis

The role of neutrophils in ischemia/reperfusion- and sepsis-induced acute kidney injury

Allicin ameliorates sepsis-induced acute kidney injury through Nrf2/HO-1 signaling pathway

Sepsis-induced myocardial dysfunction: pathophysiology and management

Gene signature for the prediction of the trajectories of sepsis-induced acute kidney injury

Sepsis-induced cardiac dysfunction and β-adrenergic blockade therapy for sepsis

PLK1 protects against sepsis-induced intestinal barrier dysfunction

Doxycycline protects against sepsis-induced endothelial glycocalyx shedding

Molecular differences in susceptibility of the kidney to sepsis-induced kidney injury

Sepsis-induced erythrocytes apoptosis

Modulating Nrf-2/HO-1, apoptosis and oxidative stress signaling pathways by gabapentin ameliorates sepsis-induced acute kidney injury

Pathophysiology of sepsis-induced myocardial dysfunction

Sepsis-induced coagulopathy (SIC) in the management of sepsis

The role of neutrophils in ischemia/reperfusion- and sepsis-induced acute kidney injury

Allicin ameliorates sepsis-induced acute kidney injury through Nrf2/HO-1 signaling pathway

Sepsis-induced myocardial dysfunction: pathophysiology and management

Gene signature for the prediction of the trajectories of sepsis-induced acute kidney injury

Sepsis-induced cardiac dysfunction and β-adrenergic blockade therapy for sepsis

PLK1 protects against sepsis-induced intestinal barrier dysfunction

Doxycycline protects against sepsis-induced endothelial glycocalyx shedding

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