Primate TRIM34 is a broadly-acting, TRIM5-dependent lentiviral restriction factor
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource, 1 online resource.
- Language
-
Englisch
- Bibliographic citation
-
Primate TRIM34 is a broadly-acting, TRIM5-dependent lentiviral restriction factor ; volume:20 ; number:1 ; day:22 ; month:8 ; year:2023 ; pages:1-14 ; date:12.2023
Retrovirology ; 20, Heft 1 (22.8.2023), 1-14, 12.2023
- Creator
-
Twentyman, Joy
Khalifeh, Anthony
Felton, Abby L.
Emerman, Michael
Ohainle, Molly
- Contributor
-
SpringerLink (Online service)
- DOI
-
10.1186/s12977-023-00629-4
- URN
-
urn:nbn:de:101:1-2023110608513917896388
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:58 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Twentyman, Joy
- Khalifeh, Anthony
- Felton, Abby L.
- Emerman, Michael
- Ohainle, Molly
- SpringerLink (Online service)
Other Objects (12)
Nef-mediated primate lentiviral immune evasion mechanisms
Regulation of primate lentiviral RNA dimerization by structural entrapment
Primate lentiviral Nef proteins deregulate T-cell development by multiple mechanisms
Hallmarks of primate lentiviral immunodeficiency infection recapitulate loss of innate lymphoid cells
PIM kinases facilitate lentiviral evasion from SAMHD1 restriction via Vpx phosphorylation
Optical coherence tomography analysis of primate retinae after lentiviral LV-RPE65 gene transfer
Species-specific host factors rather than virus-intrinsic virulence determine primate lentiviral pathogenicity
Potent and reversible lentiviral vector restriction in murine induced pluripotent stem cells
Good CoP, bad CoP? Interrogating the immune responses to primate lentiviral vaccines
Correction to: Potent and reversible lentiviral vector restriction in murine induced pluripotent stem cells
Modulation of viral gene expression and cellular immune responses by primate lentiviral proteins Vpu, Vpr and Nef
Primate Report
Nef-mediated primate lentiviral immune evasion mechanisms
Regulation of primate lentiviral RNA dimerization by structural entrapment
Primate lentiviral Nef proteins deregulate T-cell development by multiple mechanisms
Hallmarks of primate lentiviral immunodeficiency infection recapitulate loss of innate lymphoid cells
PIM kinases facilitate lentiviral evasion from SAMHD1 restriction via Vpx phosphorylation
Optical coherence tomography analysis of primate retinae after lentiviral LV-RPE65 gene transfer
Species-specific host factors rather than virus-intrinsic virulence determine primate lentiviral pathogenicity
Potent and reversible lentiviral vector restriction in murine induced pluripotent stem cells
Good CoP, bad CoP? Interrogating the immune responses to primate lentiviral vaccines
Correction to: Potent and reversible lentiviral vector restriction in murine induced pluripotent stem cells
Modulation of viral gene expression and cellular immune responses by primate lentiviral proteins Vpu, Vpr and Nef
Primate Report
Nef-mediated primate lentiviral immune evasion mechanisms
Regulation of primate lentiviral RNA dimerization by structural entrapment
Primate lentiviral Nef proteins deregulate T-cell development by multiple mechanisms
Hallmarks of primate lentiviral immunodeficiency infection recapitulate loss of innate lymphoid cells
PIM kinases facilitate lentiviral evasion from SAMHD1 restriction via Vpx phosphorylation
Optical coherence tomography analysis of primate retinae after lentiviral LV-RPE65 gene transfer
Species-specific host factors rather than virus-intrinsic virulence determine primate lentiviral pathogenicity
Potent and reversible lentiviral vector restriction in murine induced pluripotent stem cells
Good CoP, bad CoP? Interrogating the immune responses to primate lentiviral vaccines
Correction to: Potent and reversible lentiviral vector restriction in murine induced pluripotent stem cells
Modulation of viral gene expression and cellular immune responses by primate lentiviral proteins Vpu, Vpr and Nef