NDST3 deacetylates α‐tubulin and suppresses V‐ATPase assembly and lysosomal acidification

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Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: Online-Ressource

Language: Englisch

Bibliographic citation: NDST3 deacetylates α‐tubulin and suppresses V‐ATPase assembly and lysosomal acidification ; day:25 ; month:08 ; year:2021 ; extent:18
The EMBO journal / European Molecular Biology Organization ; (25.08.2021) (gesamt 18)

Creator: Tang, Qing
Liu, Mingming
Liu, Yang
Hwang, Ran‐Der
Zhang, Tao
Wang, Jiou

DOI: 10.15252/embj.2020107204

URN: urn:nbn:de:101:1-2021082615055429203587

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:56 AM CEST

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Tang, Qing
Liu, Mingming
Liu, Yang
Hwang, Ran‐Der
Zhang, Tao
Wang, Jiou

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NDST3 deacetylates α‐tubulin and suppresses V‐ATPase assembly and lysosomal acidification

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Other Objects (12)

Defective Lamtor5 Leads to Autoimmunity by Deregulating v‐ATPase and Lysosomal Acidification

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Mitochondrial translation deficiency impairs NAD + ‐mediated lysosomal acidification

Prosapogenin A induces GSDME-dependent pyroptosis of anaplastic thyroid cancer through vacuolar ATPase activation-mediated lysosomal over-acidification

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Lysosomal acidification dysfunction in microglia: an emerging pathogenic mechanism of neuroinflammation and neurodegeneration

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Defective Lamtor5 Leads to Autoimmunity by Deregulating v‐ATPase and Lysosomal Acidification

The emerging roles of vacuolar-type ATPase-dependent Lysosomal acidification in neurodegenerative diseases

Leucine/glutamine and v-ATPase/lysosomal acidification via mTORC1 activation are required for position-dependent regeneration

Mitochondrial translation deficiency impairs NAD + ‐mediated lysosomal acidification

Prosapogenin A induces GSDME-dependent pyroptosis of anaplastic thyroid cancer through vacuolar ATPase activation-mediated lysosomal over-acidification

mTORC1 regulates lysosomal catabolic activity by preventing V-ATPase assembly

Defective lysosomal acidification: a new prognostic marker and therapeutic target for neurodegenerative diseases

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