Nuclear exclusion of YAP exacerbates podocyte apoptosis and disease progression in Adriamycin-induced focal segmental glomerulosclerosis
- Standort
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1530-0307
- Umfang
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Online-Ressource
- Sprache
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Englisch
- Anmerkungen
-
online resource.
- Erschienen in
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Nuclear exclusion of YAP exacerbates podocyte apoptosis and disease progression in Adriamycin-induced focal segmental glomerulosclerosis ; day:17 ; month:11 ; year:2020 ; pages:1-13
Laboratory investigation ; (17.11.2020), 1-13
- Urheber
-
Zhuang, Qiyuan
Li, Fang
Liu, Jun
Wang, Hongyu
Tian, Yuchen
Zhang, Zhigang
Wang, Feng
Zhao, Zhonghua
Chen, Jianchun
Wu, Huijuan
- Beteiligte Personen und Organisationen
-
SpringerLink (Online service)
- DOI
-
10.1038/s41374-020-00503-3
- URN
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urn:nbn:de:101:1-2020122820451620780561
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
15.08.2025, 07:38 MESZ
Datenpartner
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Beteiligte
- Zhuang, Qiyuan
- Li, Fang
- Liu, Jun
- Wang, Hongyu
- Tian, Yuchen
- Zhang, Zhigang
- Wang, Feng
- Zhao, Zhonghua
- Chen, Jianchun
- Wu, Huijuan
- SpringerLink (Online service)
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Pharmacogenetic podocyte depletion in zebrafish larvae as a model for focal segmental glomerulosclerosis
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Acatalasemic mice are mildly susceptible to adriamycin nephropathy and exhibit increased albuminuria and glomerulosclerosis
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B7-1 mediates podocyte injury and glomerulosclerosis through communication with Hsp90ab1-LRP5-β-catenin pathway
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Btg2 Promotes Focal Segmental Glomerulosclerosis via Smad3‐Dependent Podocyte‐Mesenchymal Transition
RhoA deficiency disrupts podocyte cytoskeleton and induces podocyte apoptosis by inhibiting YAP/dendrin signal
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Cyclosporine A Protects Podocytes by Regulating Transgelin in Adriamycin-Induced Podocyte Injury
Pharmacogenetic podocyte depletion in zebrafish larvae as a model for focal segmental glomerulosclerosis
AF1q Contributes to Adriamycin-Induced Podocyte Injury by Activating Wnt/β-Catenin Signaling
Acatalasemic mice are mildly susceptible to adriamycin nephropathy and exhibit increased albuminuria and glomerulosclerosis
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B7-1 mediates podocyte injury and glomerulosclerosis through communication with Hsp90ab1-LRP5-β-catenin pathway