LincRNA-p21 Inhibits Cisplatin-Induced Apoptosis of Human Renal Proximal Tubular Epithelial Cells by Sponging miR-449a
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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Online-Ressource
- Language
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Englisch
- Bibliographic citation
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LincRNA-p21 Inhibits Cisplatin-Induced Apoptosis of Human Renal Proximal Tubular Epithelial Cells by Sponging miR-449a ; volume:46 ; number:4 ; year:2021 ; pages:495-501 ; extent:7
Kidney & blood pressure research ; 46, Heft 4 (2021), 495-501 (gesamt 7)
- Creator
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Li, Zhen
Hou, Gang
- DOI
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10.1159/000509229
- URN
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urn:nbn:de:101:1-2021090200154406958239
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:29 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Li, Zhen
- Hou, Gang
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LincRNA-p21/AIF-1/CMPK2/NLRP3 pathway promoted inflammation, autophagy and apoptosis of human tubular epithelial cell induced by urate via exosomes
PARVB deficiency alleviates cisplatin-induced tubular injury by inhibiting TAK1 signaling
LincRNA-p21 promotes mesenchymal stem cell migration capacity and survival through hypoxic preconditioning
LINC_00355 promotes gastric cancer progression by upregulating PHF19 expression through sponging miR-15a-5p
LincRNAFEZF1-AS1 represses p21 expression to promote gastric cancer proliferation through LSD1-Mediated H3K4me2 demethylation
PSTPIP2 inhibits cisplatin-induced acute kidney injury by suppressing apoptosis of renal tubular epithelial cells
A lincRNA-p21/miR-181 family feedback loop regulates microglial activation during systemic LPS- and MPTP- induced neuroinflammation
Histone deacetylase inhibitors protect against cisplatin-induced acute kidney injury by activating autophagy in proximal tubular cells
Reversal of the renal hyperglycemic memory in diabetic kidney disease by targeting sustained tubular p21 expression
In vitro study on effect of bardoxolone methyl on cisplatin-induced cellular senescence in human proximal tubular cells
Penicilliumin B Protects against Cisplatin-Induced Renal Tubular Cell Apoptosis through Activation of AMPK-Induced Autophagy and Mitochondrial Biogenesis
p53-targeted lincRNA-p21 acts as a tumor suppressor by inhibiting JAK2/STAT3 signaling pathways in head and neck squamous cell carcinoma
LincRNA-p21/AIF-1/CMPK2/NLRP3 pathway promoted inflammation, autophagy and apoptosis of human tubular epithelial cell induced by urate via exosomes
PARVB deficiency alleviates cisplatin-induced tubular injury by inhibiting TAK1 signaling
LincRNA-p21 promotes mesenchymal stem cell migration capacity and survival through hypoxic preconditioning
LINC_00355 promotes gastric cancer progression by upregulating PHF19 expression through sponging miR-15a-5p
LincRNAFEZF1-AS1 represses p21 expression to promote gastric cancer proliferation through LSD1-Mediated H3K4me2 demethylation
PSTPIP2 inhibits cisplatin-induced acute kidney injury by suppressing apoptosis of renal tubular epithelial cells
A lincRNA-p21/miR-181 family feedback loop regulates microglial activation during systemic LPS- and MPTP- induced neuroinflammation
Histone deacetylase inhibitors protect against cisplatin-induced acute kidney injury by activating autophagy in proximal tubular cells
Reversal of the renal hyperglycemic memory in diabetic kidney disease by targeting sustained tubular p21 expression
In vitro study on effect of bardoxolone methyl on cisplatin-induced cellular senescence in human proximal tubular cells
Penicilliumin B Protects against Cisplatin-Induced Renal Tubular Cell Apoptosis through Activation of AMPK-Induced Autophagy and Mitochondrial Biogenesis