Primary glia expressing the G93A-SOD1 mutation present a neuroinflammatory phenotype and provide a cellular system for studies of glial inflammation
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1742-2094
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Primary glia expressing the G93A-SOD1 mutation present a neuroinflammatory phenotype and provide a cellular system for studies of glial inflammation ; volume:3 ; number:1 ; day:25 ; month:1 ; year:2006 ; pages:1-9 ; date:12.2006
Journal of neuroinflammation ; 3, Heft 1 (25.1.2006), 1-9, 12.2006
- Creator
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Hensley, Kenneth
- Contributor
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Abdel-Moaty, Haitham
Hunter, Jerrod
Mhatre, Molina
Mou, Shenyun
Nguyen, Kim
Potapova, Tamara
Pye, Quentin N.
Qi, Min
Rice, Heather
Stewart, Charles
Stroukoff, Katharine
West, Melinda
SpringerLink (Online service)
- DOI
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10.1186/1742-2094-3-2
- URN
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urn:nbn:de:1111-2016032325770
- Rights
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Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:59 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Hensley, Kenneth
- Abdel-Moaty, Haitham
- Hunter, Jerrod
- Mhatre, Molina
- Mou, Shenyun
- Nguyen, Kim
- Potapova, Tamara
- Pye, Quentin N.
- Qi, Min
- Rice, Heather
- Stewart, Charles
- Stroukoff, Katharine
- West, Melinda
- SpringerLink (Online service)
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Neuron-glia interaction controlling glial migration in drosophila
Glial-neuronal signaling mechanisms underlying the neuroinflammatory effects of manganese
Cortical glia in SOD1(G93A) mice are subtly affected by ALS-like pathology
Signaling between neurons and NG2 glia : glial signal integration and protein synthesis
RNS60 exerts therapeutic effects in the SOD1 ALS mouse model through protective glia and peripheral nerve rescue
Neuron-glia communication via EphA4-ephrinA3 modulates LTP through glial glutamate transport
Cells Expressing Prominin-1 in Neonatal Murine Inferior Colliculus Differentiate into Neurons and Glia
Skeletal myotubes expressing ALS mutant SOD1 induce pathogenic changes, impair mitochondrial axonal transport, and trigger motoneuron death
Young glial progenitor cells competitively replace aged and diseased human glia in the adult chimeric mouse brain
Migraine: a neuroinflammatory disease?
Der Einfluss einer wildtypischen SOD1-Untereinheit auf Konformation und Stabilität mutanter SOD1-Heterodimere
Der Einfluss einer wildtypischen SOD1-Untereinheit auf Konformation und Stabilität mutanter SOD1-Heterodimere
Neuron-glia interaction controlling glial migration in drosophila
Glial-neuronal signaling mechanisms underlying the neuroinflammatory effects of manganese
Cortical glia in SOD1(G93A) mice are subtly affected by ALS-like pathology
Signaling between neurons and NG2 glia : glial signal integration and protein synthesis
RNS60 exerts therapeutic effects in the SOD1 ALS mouse model through protective glia and peripheral nerve rescue
Neuron-glia communication via EphA4-ephrinA3 modulates LTP through glial glutamate transport
Cells Expressing Prominin-1 in Neonatal Murine Inferior Colliculus Differentiate into Neurons and Glia
Skeletal myotubes expressing ALS mutant SOD1 induce pathogenic changes, impair mitochondrial axonal transport, and trigger motoneuron death
Young glial progenitor cells competitively replace aged and diseased human glia in the adult chimeric mouse brain
Migraine: a neuroinflammatory disease?
Der Einfluss einer wildtypischen SOD1-Untereinheit auf Konformation und Stabilität mutanter SOD1-Heterodimere