Suppression of TNBC metastasis by doxazosin, a novel dual inhibitor of c-MET/EGFR

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Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: 1 Online-Ressource.

Language: Englisch

Bibliographic citation: Suppression of TNBC metastasis by doxazosin, a novel dual inhibitor of c-MET/EGFR ; volume:42 ; number:1 ; day:4 ; month:11 ; year:2023 ; pages:1-19 ; date:12.2023
Journal of experimental & clinical cancer research ; 42, Heft 1 (4.11.2023), 1-19, 12.2023

Creator: Kim, Seongjae
Park, Jung Min
Park, Soeun
Jung, Eunsun
Ko, Dongmi
Park, Minsu
Seo, Juyeon
Nam, Kee Dal
Kang, Yong Koo
Lee, Kyoungmin
Farrand, Lee
Kim, Yoon-Jae
Kim, Ji Young
Seo, Jae Hong

Contributor: SpringerLink (Online service)

DOI: 10.1186/s13046-023-02866-z

URN: urn:nbn:de:101:1-2024012213231704705911

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:25 AM CEST

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Associated

Kim, Seongjae
Park, Jung Min
Park, Soeun
Jung, Eunsun
Ko, Dongmi
Park, Minsu
Seo, Juyeon
Nam, Kee Dal
Kang, Yong Koo
Lee, Kyoungmin
Farrand, Lee
Kim, Yoon-Jae
Kim, Ji Young
Seo, Jae Hong
SpringerLink (Online service)

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PDZK1 suppresses TNBC development and sensitizes TNBC cells to erlotinib via the EGFR pathway

DUSP22 inhibits lung tumorigenesis by suppression of EGFR/c-Met signaling

EZH2-H3K27me3 mediated KRT14 upregulation promotes TNBC peritoneal metastasis

GluOC promotes proliferation and metastasis of TNBC through the ROCK1 signaling pathway

Lycorine inhibits glioblastoma multiforme growth through EGFR suppression

Hochschulschrift

HER2/neu, EGFR und c-Met-Mutation/ Amplifikation

ASAH1 facilitates TNBC by DUSP5 suppression-driven activation of MAP kinase pathway and represents a therapeutic vulnerability

Brain metastasis, EGFR mutation subtype and generation of EGFR-TKI jointly influence the treatment outcome of patient with EGFR-mutant NSCLC

The incorporation of acetylated LAP-TGF-β1 proteins into exosomes promotes TNBC cell dissemination in lung micro-metastasis

Cell morphology best predicts tumorigenicity and metastasis in vivo across multiple TNBC cell lines of different metastatic potential

Hochschulschrift

c-Met als prognostischer Biomarker im EGFR-mutierten nichtkleinzelligen Lungenkarzinom

Growth suppression of colorectal cancer expressing S492R EGFR by monoclonal antibody CH12

PDZK1 suppresses TNBC development and sensitizes TNBC cells to erlotinib via the EGFR pathway

DUSP22 inhibits lung tumorigenesis by suppression of EGFR/c-Met signaling

EZH2-H3K27me3 mediated KRT14 upregulation promotes TNBC peritoneal metastasis

GluOC promotes proliferation and metastasis of TNBC through the ROCK1 signaling pathway

Lycorine inhibits glioblastoma multiforme growth through EGFR suppression

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HER2/neu, EGFR und c-Met-Mutation/ Amplifikation

ASAH1 facilitates TNBC by DUSP5 suppression-driven activation of MAP kinase pathway and represents a therapeutic vulnerability

Brain metastasis, EGFR mutation subtype and generation of EGFR-TKI jointly influence the treatment outcome of patient with EGFR-mutant NSCLC

The incorporation of acetylated LAP-TGF-β1 proteins into exosomes promotes TNBC cell dissemination in lung micro-metastasis

Cell morphology best predicts tumorigenicity and metastasis in vivo across multiple TNBC cell lines of different metastatic potential

Hochschulschrift

c-Met als prognostischer Biomarker im EGFR-mutierten nichtkleinzelligen Lungenkarzinom

Growth suppression of colorectal cancer expressing S492R EGFR by monoclonal antibody CH12

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Suppression of TNBC metastasis by doxazosin, a novel dual inhibitor of c-MET/EGFR

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Other Objects (12)

PDZK1 suppresses TNBC development and sensitizes TNBC cells to erlotinib via the EGFR pathway

DUSP22 inhibits lung tumorigenesis by suppression of EGFR/c-Met signaling

EZH2-H3K27me3 mediated KRT14 upregulation promotes TNBC peritoneal metastasis

GluOC promotes proliferation and metastasis of TNBC through the ROCK1 signaling pathway

Lycorine inhibits glioblastoma multiforme growth through EGFR suppression

HER2/neu, EGFR und c-Met-Mutation/ Amplifikation

ASAH1 facilitates TNBC by DUSP5 suppression-driven activation of MAP kinase pathway and represents a therapeutic vulnerability

Brain metastasis, EGFR mutation subtype and generation of EGFR-TKI jointly influence the treatment outcome of patient with EGFR-mutant NSCLC

The incorporation of acetylated LAP-TGF-β1 proteins into exosomes promotes TNBC cell dissemination in lung micro-metastasis

Cell morphology best predicts tumorigenicity and metastasis in vivo across multiple TNBC cell lines of different metastatic potential

c-Met als prognostischer Biomarker im EGFR-mutierten nichtkleinzelligen Lungenkarzinom

Growth suppression of colorectal cancer expressing S492R EGFR by monoclonal antibody CH12

PDZK1 suppresses TNBC development and sensitizes TNBC cells to erlotinib via the EGFR pathway

DUSP22 inhibits lung tumorigenesis by suppression of EGFR/c-Met signaling

EZH2-H3K27me3 mediated KRT14 upregulation promotes TNBC peritoneal metastasis

GluOC promotes proliferation and metastasis of TNBC through the ROCK1 signaling pathway

Lycorine inhibits glioblastoma multiforme growth through EGFR suppression

HER2/neu, EGFR und c-Met-Mutation/ Amplifikation

ASAH1 facilitates TNBC by DUSP5 suppression-driven activation of MAP kinase pathway and represents a therapeutic vulnerability

Brain metastasis, EGFR mutation subtype and generation of EGFR-TKI jointly influence the treatment outcome of patient with EGFR-mutant NSCLC

The incorporation of acetylated LAP-TGF-β1 proteins into exosomes promotes TNBC cell dissemination in lung micro-metastasis

Cell morphology best predicts tumorigenicity and metastasis in vivo across multiple TNBC cell lines of different metastatic potential

c-Met als prognostischer Biomarker im EGFR-mutierten nichtkleinzelligen Lungenkarzinom

Growth suppression of colorectal cancer expressing S492R EGFR by monoclonal antibody CH12

PDZK1 suppresses TNBC development and sensitizes TNBC cells to erlotinib via the EGFR pathway

DUSP22 inhibits lung tumorigenesis by suppression of EGFR/c-Met signaling

EZH2-H3K27me3 mediated KRT14 upregulation promotes TNBC peritoneal metastasis

GluOC promotes proliferation and metastasis of TNBC through the ROCK1 signaling pathway

Lycorine inhibits glioblastoma multiforme growth through EGFR suppression

HER2/neu, EGFR und c-Met-Mutation/ Amplifikation

ASAH1 facilitates TNBC by DUSP5 suppression-driven activation of MAP kinase pathway and represents a therapeutic vulnerability

Brain metastasis, EGFR mutation subtype and generation of EGFR-TKI jointly influence the treatment outcome of patient with EGFR-mutant NSCLC

The incorporation of acetylated LAP-TGF-β1 proteins into exosomes promotes TNBC cell dissemination in lung micro-metastasis

Cell morphology best predicts tumorigenicity and metastasis in vivo across multiple TNBC cell lines of different metastatic potential

c-Met als prognostischer Biomarker im EGFR-mutierten nichtkleinzelligen Lungenkarzinom

Growth suppression of colorectal cancer expressing S492R EGFR by monoclonal antibody CH12

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