EMT and stemness: flexible processes tuned by alternative splicing in development and cancer progression
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1476-4598
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
EMT and stemness: flexible processes tuned by alternative splicing in development and cancer progression ; volume:16 ; number:1 ; day:30 ; month:1 ; year:2017 ; pages:1-19 ; date:12.2017
Molecular cancer ; 16, Heft 1 (30.1.2017), 1-19, 12.2017
- Keyword
-
Krebs
Onkologie
- Creator
-
Pradella, Davide
- Contributor
-
Naro, Chiara
Sette, Claudio
Ghigna, Claudia
SpringerLink (Online service)
- DOI
-
10.1186/s12943-016-0579-2
- URN
-
urn:nbn:de:1111-2017021110666
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:52 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Pradella, Davide
- Naro, Chiara
- Sette, Claudio
- Ghigna, Claudia
- SpringerLink (Online service)
Other Objects (12)
Inhibitory role of ATF3 in gastric cancer progression through regulating cell EMT and stemness
miR-200 is a potent regulator of cancer progression by repressing EMT and stemness
The ZEB1-miR-200 feedback loop controls the Notch signaling pathway and alternative splicing to regulate EMT and stemness
Chemotherapeutic stress influences EMT and stemness in cancer stem cells of TNBC
Loss of Stemness, EMT, and Supernumerary Tooth Formation in Cebpb −/− Runx2 +/− Murine Incisors
How BMP-2 induces EMT and breast cancer stemness through Rb and CD44?
Pancreatic cancer stemness: dynamic status in malignant progression
Dynamic EMT: a multi‐tool for tumor progression
The dynamic interplay of ZEB1, CD44 and HAS2 promotes EMT and stemness in cancer
Alternative splicing associated with cancer stemness in kidney renal clear cell carcinoma
The EMT inducer SNAIL1 deregulates the expression of stemness-associated genes in colorectal cancer cells
PI3Kδ activity controls plasticity and discriminates between EMT and stemness based on distinct TGFβ signaling
Inhibitory role of ATF3 in gastric cancer progression through regulating cell EMT and stemness
miR-200 is a potent regulator of cancer progression by repressing EMT and stemness
The ZEB1-miR-200 feedback loop controls the Notch signaling pathway and alternative splicing to regulate EMT and stemness
Chemotherapeutic stress influences EMT and stemness in cancer stem cells of TNBC
Loss of Stemness, EMT, and Supernumerary Tooth Formation in Cebpb −/− Runx2 +/− Murine Incisors
How BMP-2 induces EMT and breast cancer stemness through Rb and CD44?
Pancreatic cancer stemness: dynamic status in malignant progression
Dynamic EMT: a multi‐tool for tumor progression
The dynamic interplay of ZEB1, CD44 and HAS2 promotes EMT and stemness in cancer
Alternative splicing associated with cancer stemness in kidney renal clear cell carcinoma
The EMT inducer SNAIL1 deregulates the expression of stemness-associated genes in colorectal cancer cells
PI3Kδ activity controls plasticity and discriminates between EMT and stemness based on distinct TGFβ signaling
Inhibitory role of ATF3 in gastric cancer progression through regulating cell EMT and stemness
miR-200 is a potent regulator of cancer progression by repressing EMT and stemness
The ZEB1-miR-200 feedback loop controls the Notch signaling pathway and alternative splicing to regulate EMT and stemness
Chemotherapeutic stress influences EMT and stemness in cancer stem cells of TNBC
Loss of Stemness, EMT, and Supernumerary Tooth Formation in Cebpb −/− Runx2 +/− Murine Incisors
How BMP-2 induces EMT and breast cancer stemness through Rb and CD44?
Pancreatic cancer stemness: dynamic status in malignant progression
Dynamic EMT: a multi‐tool for tumor progression
The dynamic interplay of ZEB1, CD44 and HAS2 promotes EMT and stemness in cancer
Alternative splicing associated with cancer stemness in kidney renal clear cell carcinoma
The EMT inducer SNAIL1 deregulates the expression of stemness-associated genes in colorectal cancer cells