MH84 improves mitochondrial dysfunction in a mouse model of early Alzheimer’s disease

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1758-9193

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: MH84 improves mitochondrial dysfunction in a mouse model of early Alzheimer’s disease ; volume:10 ; number:1 ; day:13 ; month:2 ; year:2018 ; pages:1-12 ; date:12.2018
Alzheimer's research & therapy ; 10, Heft 1 (13.2.2018), 1-12, 12.2018

Keyword: Biopharmazie
Pharmakodynamik
Pharmakokinetik

Creator: Pohland, Maximilian

Contributor: Pellowska, Maren
Asseburg, Heike
Hagl, Stephanie
Reutzel, Martina
Joppe, Aljoscha
Berressem, Dirk
Eckert, Schamim H.
Wurglics, Mario
Schubert‐Zsilavecz, Manfred
Eckert, Gunter P.
SpringerLink (Online service)

DOI: 10.1186/s13195-018-0342-6

URN: urn:nbn:de:1111-201804057729

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 11:02 AM CEST

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Associated

Pohland, Maximilian
Pellowska, Maren
Asseburg, Heike
Hagl, Stephanie
Reutzel, Martina
Joppe, Aljoscha
Berressem, Dirk
Eckert, Schamim H.
Wurglics, Mario
Schubert‐Zsilavecz, Manfred
Eckert, Gunter P.
SpringerLink (Online service)

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MH84 improves mitochondrial dysfunction in a mouse model of early Alzheimer’s disease

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Other Objects (12)

MH84 improves mitochondrial dysfunction in a mouse model of early Alzheimer’s disease

AIBP controls TLR4 inflammarafts and mitochondrial dysfunction in a mouse model of Alzheimer’s disease

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Targeting the overexpressed mitochondrial protein VDAC1 in a mouse model of Alzheimer’s disease protects against mitochondrial dysfunction and mitigates brain pathology

Mitochondrial alterations in different Alzheimers disease mouse models

Mitochondrial dysfunction in microglia: a novel perspective for pathogenesis of Alzheimer’s disease

Nitric oxide donors rescue metabolic and mitochondrial dysfunction in obese Alzheimer’s model

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Modulation of astrocyte reactivity improves functional deficits in mouse models of Alzheimer’s disease

MH84 improves mitochondrial dysfunction in a mouse model of early Alzheimer’s disease

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Targeting the overexpressed mitochondrial protein VDAC1 in a mouse model of Alzheimer’s disease protects against mitochondrial dysfunction and mitigates brain pathology

Mitochondrial alterations in different Alzheimers disease mouse models

Mitochondrial dysfunction in microglia: a novel perspective for pathogenesis of Alzheimer’s disease

Nitric oxide donors rescue metabolic and mitochondrial dysfunction in obese Alzheimer’s model

Correction: Targeting the overexpressed mitochondrial protein VDAC1 in a mouse model of Alzheimer’s disease protects against mitochondrial dysfunction and mitigates brain pathology

ACSS2-dependent histone acetylation improves cognition in mouse model of Alzheimer’s disease

Mitochondrial Alterations in Alzheimer’s Disease: Insight from the 5xFAD Mouse Model

Diastolic dysfunction in Alzheimer’s disease model mice is associated with Aβ-amyloid aggregate formation and mitochondrial dysfunction

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Targeting the overexpressed mitochondrial protein VDAC1 in a mouse model of Alzheimer’s disease protects against mitochondrial dysfunction and mitigates brain pathology

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