Correction to: Loss of homeostatic microglial phenotype in CSF1R-related Leukoencephalopathy

to related objects

Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 2051-5960

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Correction to: Loss of homeostatic microglial phenotype in CSF1R-related Leukoencephalopathy ; volume:8 ; number:1 ; day:24 ; month:6 ; year:2020 ; pages:1-2 ; date:12.2020
Acta Neuropathologica Communications ; 8, Heft 1 (24.6.2020), 1-2, 12.2020

Creator: Kempthorne, Liam
Yoon, Hyejin
Madore, Charlotte
Smith, Scott
Wszolek, Zbigniew K.
Rademakers, Rosa
Kim, Jungsu
Butovsky, Oleg
Dickson, Dennis W.

Contributor: SpringerLink (Online service)

DOI: 10.1186/s40478-020-00970-1

URN: urn:nbn:de:101:1-2020082022400570575756

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:47 AM CEST

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Associated

Kempthorne, Liam
Yoon, Hyejin
Madore, Charlotte
Smith, Scott
Wszolek, Zbigniew K.
Rademakers, Rosa
Kim, Jungsu
Butovsky, Oleg
Dickson, Dennis W.
SpringerLink (Online service)

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Correction to: Loss of homeostatic microglial phenotype in CSF1R-related Leukoencephalopathy

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Other Objects (12)

Loss of homeostatic microglial phenotype in CSF1R-related Leukoencephalopathy

The neuropathological diagnosis of Alzheimer’s disease

Cellular and pathological heterogeneity of primary tauopathies

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Neuropathology and molecular diagnosis of Synucleinopathies

Proaggregant nuclear factor(s) trigger rapid formation of α-synuclein aggregates in apoptotic neurons

TDP-43 Pathology in Alzheimer’s Disease

C-terminal and full length TDP-43 specie differ according to FTLD-TDP lesion type but not genetic mutation

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The neuropathological diagnosis of Alzheimer’s disease

Cellular and pathological heterogeneity of primary tauopathies

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Proaggregant nuclear factor(s) trigger rapid formation of α-synuclein aggregates in apoptotic neurons

TDP-43 Pathology in Alzheimer’s Disease

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Neuropathology and molecular diagnosis of Synucleinopathies

Proaggregant nuclear factor(s) trigger rapid formation of α-synuclein aggregates in apoptotic neurons

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C-terminal and full length TDP-43 specie differ according to FTLD-TDP lesion type but not genetic mutation

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Expression and processing analyses of wild type and p.R47H TREM2 variant in Alzheimer’s disease brains

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