Impact of klotho on the expression of SRGAP2a in podocytes in diabetic nephropathy
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1471-2369
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
-
Impact of klotho on the expression of SRGAP2a in podocytes in diabetic nephropathy ; volume:23 ; number:1 ; day:18 ; month:4 ; year:2022 ; pages:1-12 ; date:12.2022
BMC nephrology ; 23, Heft 1 (18.4.2022), 1-12, 12.2022
- Creator
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Jin, Donghua
Jia, Miao
Xie, Yuxian
Lin, Lihua
Qiu, Hong
Lu, Guoyuan
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/s12882-022-02765-z
- URN
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urn:nbn:de:101:1-2022070207010838151278
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:31 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Jin, Donghua
- Jia, Miao
- Xie, Yuxian
- Lin, Lihua
- Qiu, Hong
- Lu, Guoyuan
- SpringerLink (Online service)
Other Objects (12)
Deletion of IRE1α in podocytes exacerbates diabetic nephropathy in mice
Spironolactone alleviates diabetic nephropathy through promoting autophagy in podocytes
METTL14 promotes glomerular endothelial cell injury and diabetic nephropathy via m6A modification of α-klotho
Retraction Note: Silencing of Histone Deacetylase 9 Expression in Podocytes Attenuates Kidney Injury in Diabetic Nephropathy
Diabetic nephropathy
Correction to: METTL14 promotes glomerular endothelial cell injury and diabetic nephropathy via m6A modification of α‑klotho
Swiprosin-1 Promotes Mitochondria-Dependent Apoptosis of Glomerular Podocytes via P38 MAPK Pathway in Early-Stage Diabetic Nephropathy
Detection of urinary podocytes by flow cytometry in idiopathic membranous nephropathy
Genetic Basis of Diabetic Nephropathy
The crosstalk between glomerular endothelial cells and podocytes controls their responses to metabolic stimuli in diabetic nephropathy
ADP-ribosylation factor-interacting protein 2 acts as a novel regulator of mitophagy and autophagy in podocytes in diabetic nephropathy
Author Correction: Detection of urinary podocytes by flow cytometry in idiopathic membranous nephropathy
Deletion of IRE1α in podocytes exacerbates diabetic nephropathy in mice
Spironolactone alleviates diabetic nephropathy through promoting autophagy in podocytes
METTL14 promotes glomerular endothelial cell injury and diabetic nephropathy via m6A modification of α-klotho
Retraction Note: Silencing of Histone Deacetylase 9 Expression in Podocytes Attenuates Kidney Injury in Diabetic Nephropathy
Diabetic nephropathy
Correction to: METTL14 promotes glomerular endothelial cell injury and diabetic nephropathy via m6A modification of α‑klotho
Swiprosin-1 Promotes Mitochondria-Dependent Apoptosis of Glomerular Podocytes via P38 MAPK Pathway in Early-Stage Diabetic Nephropathy
Detection of urinary podocytes by flow cytometry in idiopathic membranous nephropathy
Genetic Basis of Diabetic Nephropathy
The crosstalk between glomerular endothelial cells and podocytes controls their responses to metabolic stimuli in diabetic nephropathy
ADP-ribosylation factor-interacting protein 2 acts as a novel regulator of mitophagy and autophagy in podocytes in diabetic nephropathy
Author Correction: Detection of urinary podocytes by flow cytometry in idiopathic membranous nephropathy
Deletion of IRE1α in podocytes exacerbates diabetic nephropathy in mice
Spironolactone alleviates diabetic nephropathy through promoting autophagy in podocytes
METTL14 promotes glomerular endothelial cell injury and diabetic nephropathy via m6A modification of α-klotho
Retraction Note: Silencing of Histone Deacetylase 9 Expression in Podocytes Attenuates Kidney Injury in Diabetic Nephropathy
Diabetic nephropathy
Correction to: METTL14 promotes glomerular endothelial cell injury and diabetic nephropathy via m6A modification of α‑klotho
Swiprosin-1 Promotes Mitochondria-Dependent Apoptosis of Glomerular Podocytes via P38 MAPK Pathway in Early-Stage Diabetic Nephropathy
Detection of urinary podocytes by flow cytometry in idiopathic membranous nephropathy
Genetic Basis of Diabetic Nephropathy
The crosstalk between glomerular endothelial cells and podocytes controls their responses to metabolic stimuli in diabetic nephropathy
ADP-ribosylation factor-interacting protein 2 acts as a novel regulator of mitophagy and autophagy in podocytes in diabetic nephropathy