Activation of Rac-1 and RhoA contributes to podocyte injury in chronic kidney disease

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Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: Online-Ressource

Language: Englisch

Bibliographic citation: In: PLoS One, volume 8, issue 11, e80328 (2013)

Keyword: Urologie
Urogenitalkrankheit
Harnwegskrankheit

Event: Veröffentlichung

(where): Frankfurt am Main

(who): Univ.-Bibliothek Frankfurt am Main

(when): 2013

Contributor: Bábelová, Andrea
Jansen, Felix
Sander, Kerstin
Löhn, Matthias
Schäfer, Liliana
Fork, Christian
Rütten, Hartmut
Plettenburg, Oliver
Stark, Holger
Daniel, Christoph
Amann, Kerstin
Pavenstädt, Hermann
Jung, Oliver
Brandes, Ralf

URN: urn:nbn:de:hebis:30:3-322510

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:31 AM CEST

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Associated

Bábelová, Andrea
Jansen, Felix
Sander, Kerstin
Löhn, Matthias
Schäfer, Liliana
Fork, Christian
Rütten, Hartmut
Plettenburg, Oliver
Stark, Holger
Daniel, Christoph
Amann, Kerstin
Pavenstädt, Hermann
Jung, Oliver
Brandes, Ralf
Univ.-Bibliothek Frankfurt am Main

Time of origin

2013

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Activation of Rac-1 and RhoA Contributes to Podocyte Injury in Chronic Kidney Disease

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Experimental uninephrectomy associates with less parasympathetic modulation of heart rate and facilitates sodium-dependent arterial hypertension

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RhoA deficiency disrupts podocyte cytoskeleton and induces podocyte apoptosis by inhibiting YAP/dendrin signal

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Activation of Rac-1 and RhoA Contributes to Podocyte Injury in Chronic Kidney Disease

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Sauerstoffradikale - Signalmoleküle in der Gefäßwand

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Europhysiology 2022: bringing together the physiologists of Europe and the World : 16–18 September 2022, Tivoli Congress Center, Copenhagen, Denmark” as subtitle

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Experimental uninephrectomy associates with less parasympathetic modulation of heart rate and facilitates sodium-dependent arterial hypertension

Sympathoactivation and rho-kinase-dependent baroreflex function in experimental renovascular hypertension with reduced kidney mass

RhoA deficiency disrupts podocyte cytoskeleton and induces podocyte apoptosis by inhibiting YAP/dendrin signal

Loss of endothelial cytochrome P450 reductase induces vascular dysfunction in mice

Activation of Rac-1 and RhoA Contributes to Podocyte Injury in Chronic Kidney Disease

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Epigenetik von Herz-Kreislauf-Erkrankungen : Forscher setzen auf "RNA-Therapeutika"

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Europhysiology 2022: bringing together the physiologists of Europe and the World : 16–18 September 2022, Tivoli Congress Center, Copenhagen, Denmark” as subtitle

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Experimental uninephrectomy associates with less parasympathetic modulation of heart rate and facilitates sodium-dependent arterial hypertension

Sympathoactivation and rho-kinase-dependent baroreflex function in experimental renovascular hypertension with reduced kidney mass

RhoA deficiency disrupts podocyte cytoskeleton and induces podocyte apoptosis by inhibiting YAP/dendrin signal

Loss of endothelial cytochrome P450 reductase induces vascular dysfunction in mice

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Activation of Rac-1 and RhoA contributes to podocyte injury in chronic kidney disease

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Other Objects (12)

Activation of Rac-1 and RhoA Contributes to Podocyte Injury in Chronic Kidney Disease

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Europhysiology 2022: bringing together the physiologists of Europe and the World : 16–18 September 2022, Tivoli Congress Center, Copenhagen, Denmark” as subtitle

Europhysiology 2022 : bringing together the physiologists of Europe and the World16–18 September 2022, Tivoli Congress Center, Copenhagen, Denmark

Experimental uninephrectomy associates with less parasympathetic modulation of heart rate and facilitates sodium-dependent arterial hypertension

Sympathoactivation and rho-kinase-dependent baroreflex function in experimental renovascular hypertension with reduced kidney mass

Sympathoactivation and rho-kinase-dependent baroreflex function in experimental renovascular hypertension with reduced kidney mass

RhoA deficiency disrupts podocyte cytoskeleton and induces podocyte apoptosis by inhibiting YAP/dendrin signal

Loss of endothelial cytochrome P450 reductase induces vascular dysfunction in mice

Activation of Rac-1 and RhoA Contributes to Podocyte Injury in Chronic Kidney Disease

Sauerstoffradikale - Signalmoleküle in der Gefäßwand

Vaskuläre Superoxidanionenproduktion : In-vitro-Untersuchungen in der lucigeninverstärkten Chemilumineszenz

Epigenetik von Herz-Kreislauf-Erkrankungen : Forscher setzen auf "RNA-Therapeutika"

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Europhysiology 2022: bringing together the physiologists of Europe and the World : 16–18 September 2022, Tivoli Congress Center, Copenhagen, Denmark” as subtitle

Europhysiology 2022 : bringing together the physiologists of Europe and the World16–18 September 2022, Tivoli Congress Center, Copenhagen, Denmark

Experimental uninephrectomy associates with less parasympathetic modulation of heart rate and facilitates sodium-dependent arterial hypertension

Sympathoactivation and rho-kinase-dependent baroreflex function in experimental renovascular hypertension with reduced kidney mass

Sympathoactivation and rho-kinase-dependent baroreflex function in experimental renovascular hypertension with reduced kidney mass

RhoA deficiency disrupts podocyte cytoskeleton and induces podocyte apoptosis by inhibiting YAP/dendrin signal

Loss of endothelial cytochrome P450 reductase induces vascular dysfunction in mice

Activation of Rac-1 and RhoA Contributes to Podocyte Injury in Chronic Kidney Disease

Sauerstoffradikale - Signalmoleküle in der Gefäßwand

Vaskuläre Superoxidanionenproduktion : In-vitro-Untersuchungen in der lucigeninverstärkten Chemilumineszenz

Epigenetik von Herz-Kreislauf-Erkrankungen : Forscher setzen auf "RNA-Therapeutika"

Schutz oder Schaden für die Gefäße? : Bei Sauerstoffradikalen kommt es auf das Gleichgewicht an [[Elektronische Ressource]]

Europhysiology 2022: bringing together the physiologists of Europe and the World : 16–18 September 2022, Tivoli Congress Center, Copenhagen, Denmark” as subtitle

Europhysiology 2022 : bringing together the physiologists of Europe and the World16–18 September 2022, Tivoli Congress Center, Copenhagen, Denmark

Experimental uninephrectomy associates with less parasympathetic modulation of heart rate and facilitates sodium-dependent arterial hypertension

Sympathoactivation and rho-kinase-dependent baroreflex function in experimental renovascular hypertension with reduced kidney mass

Sympathoactivation and rho-kinase-dependent baroreflex function in experimental renovascular hypertension with reduced kidney mass

RhoA deficiency disrupts podocyte cytoskeleton and induces podocyte apoptosis by inhibiting YAP/dendrin signal

Loss of endothelial cytochrome P450 reductase induces vascular dysfunction in mice

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